Alcohol Use of African Americans and Whites From Ages 9–20: Descriptive Results From a Longitudinal Study

Duncan, Susan C.; Strycker, Lisa A.; Duncan, Terry E.

doi:10.1080/15332640.2012.701550

Cited by 16 publications

(14 citation statements)

References 20 publications

(20 reference statements)

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“…The associations between covariates and latent growth parameters closely corresponded with prior research indicating that older adolescents drink more than younger adolescents , White youth drink more than non-White youth (Duncan et al, 2012;Fuller-Thomson et al, 2013), and Hispanic youth drink less than non-Hispanic White youth (Johnston et al, 2011;Swaim et al, 2004). In addition, supporting Problem Behavior Theory (Jessor & Jessor, 1977), use of other substances like marijuana or tobacco was associated with increased levels of alcohol use.…”

Section: Discussionsupporting

confidence: 80%

Trajectories of Adolescent Alcohol Use in the Year Following a Brief Alcohol Intervention

Abar¹,

Hernández²,

Rodríguez³

et al. 2015

J. Stud. Alcohol Drugs

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ABSTRACT. Objective: Brief interventions have become increasingly popular for youth who engage in problematic drinking behavior. The purpose of this study was to examine the alcohol use trajectories of adolescents over a 12-month period following the receipt of a brief intervention. Method: The current sample came from two independent studies and consisted of 206 adolescents (ages 13-19; 52% male) recruited through an emergency department or community institution (e.g., courts, schools). Timeline followback methods were used at four points over 1 year to obtain daily estimates of alcohol use behaviors, with daily data then aggregated at the monthly level to examine trajectories of total drinks consumed and maximum drinks on one occasion. Using latent growth curve analysis, we expected a general pattern of increasing use over time, with lower use during the month immediately following completion of the intervention. Results: Models with random intercepts, random linear slopes, and fixed quadratic trends provided good fit to the data for both total drinks and maximum drinks. For each outcome, there was an immediate decrease and then a gradual increase up to the 3-and 6-month assessments, with decreases seen in the months following assessments. Older age, White race, non-Hispanic ethnicity, and greater prior substance use were associated with greater initial levels of use and growth over time. Conclusions: Interindividual differences were observed in alcohol use trajectories over time for high-risk adolescents following an alcohol use intervention. Subsequent research may demonstrate more uniform and permanent modification of trajectories by incorporating intervention-related materials into follow-up contacts. (J. Stud. Alcohol Drugs, 76, 710-720, 2015)

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Section: Discussionsupporting

confidence: 80%

Trajectories of Adolescent Alcohol Use in the Year Following a Brief Alcohol Intervention

Abar¹,

Hernández²,

Rodríguez³

et al. 2015

J. Stud. Alcohol Drugs

View full text Add to dashboard Cite

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“…However, it is recognized that selective anatomic sites for HPV 16 infection, a sign of mucotropism occurs in concert with sexually transmission among young Caucasian males [2,30]. This group also demonstrates a unique alcohol behavior that is designated as binge drinking but it is identified with low blood alcohol concentration (BAC) and accompanied by periodic spikes of AA synthesis [31][32][33]. We suggest that this behavior pattern aids in opportunistic Streptococcus spp.…”

Section: Introductionmentioning

confidence: 92%

“…proliferation following low level (e.g., 5%,v/v, 50 mg/mL.) ETOH treatment will increase expression of Annexin A2, and EGFR to also enhance HPV 16 entry [13,14,[18][19][20][21][22][23][24][25][26][27][28][29][30][31][32].…”

Section: Set Of Comparative Assays To Examine Associations Of S Salimentioning

confidence: 99%

“…fast ADH1B*, ADH1C*1, and ALDH2*2 genotypes that result in accumulation of AA in comparison to slow ADH1B*1/1, ADH1C*1/2 genotypes and almost all Caucasians carry an additional ALDH2*1/1 genotype) [17,31].…”

Section: Introductionmentioning

confidence: 99%

“…We suggest that this behavior pattern aids in opportunistic Streptococcus spp. proliferation and HPV 16 interactions leading to enhanced risk for OPC in contrast to a lower incidence of HPV 16 related OPC among a comparable female population [31,35,36].…”

Section: Introductionmentioning

confidence: 99%

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Alcohol Treatment of Oral Streptococcus Spp. Increased the Entry of Human Papillomavirus Type 16 into Non-Malignant and Oral Squamous Cell Carcinoma Cells

2014

J Oral Biol

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Background: Oropharyngeal carcinoma is often associated with human papilloma virus subtype 16 (HPV) infections; however events that lead to HPV entry and carcinoma are poorly understood. We simulated a clinical situation in the laboratory by examining human epithelial and oral keratinocyte (HOK) responses to oral commensal bacteria (Streptococcus spp.) and metabolism of ethyl alcohol (ETOH). These studies led us to conclude that oral bacteria and ethyl alcohol through keratinocyte membrane signals act as co-factors for HPV 16 entry. Methods:We tested ETOH exposure responses of Streptococcus spp., ETOH-sensitive, acetaldehyde (AA) producers: S. mutans (LT11), S. salivarius strain, 109-2 and S. gordonii (V2016; wild type). In comparison to ETOH-resistant, non-producers of AA: S. salivarius strain, 101-7 or S. gordonii (adh ABE mutant). 5×105 per cfu/mL of bacteria were exposed to ETOH (5%,v/v) for 1h and then co-incubated for 3h with epithelial targets: immortalized: 293TT; HPV 16 (HOK/HPV 16B cells), or telomerase: hTERT HOK. Streptococci spp. attachment to epithelial cells and subsequent effects were enumerated with immune cytochemistry; Western immunoblotting, and immunoprecipitation techniques. These methods delineated expressions of syndecans-1, 4; phospho-L-tyrosine epidermal growth factor receptor (EGFR), and furin convertase (FC) with HPV 16 pseudo virus (PsV) entry into 293TT, hTERT HOKs and oral squamous cell carcinoma (SCC-25). To verify these relationships we used inhibitors of alcohol dehydrogenase (ADH), aldehyde dehydrogenase (ALDH), and FC.Results: Streptococcus spp. metabolism of ETOH released AA to trigger HOK expressions of syndecans 1 and 4 and heparin sulfate binding proteins were degraded by heparanase. Following these events enhanced phosphorylated EGFR expression and HPV 16 entry through FC activity were recorded.Conclusions: Some oral Streptococcus spp. response to ETOH mediates epithelial cell susceptibility to HPV 16 entry. This occurs through membrane associated expression changes of proteoglycans and EGFR. Moreover, oral cancer cell differentiation and growth may be dependent upon these associations.

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The relationship between childhood trauma and alcohol use initiation in Black and White adolescent girls: considering socioeconomic status and neighborhood factors

Sartor

Bachrach

Stepp

et al. 2017

Soc Psychiatry Psychiatr Epidemiol

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Alcohol Use of African Americans and Whites From Ages 9–20: Descriptive Results From a Longitudinal Study

Cited by 16 publications

References 20 publications

Trajectories of Adolescent Alcohol Use in the Year Following a Brief Alcohol Intervention

Trajectories of Adolescent Alcohol Use in the Year Following a Brief Alcohol Intervention

Alcohol Treatment of Oral Streptococcus Spp. Increased the Entry of Human Papillomavirus Type 16 into Non-Malignant and Oral Squamous Cell Carcinoma Cells

The relationship between childhood trauma and alcohol use initiation in Black and White adolescent girls: considering socioeconomic status and neighborhood factors

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