Alcohol consumption—A risk factor for hemorrhagic and non-hemorrhagic stroke

Gill, Jaswinder; Shipley, Martin J.; Tsementzis, S A; Hornby, Robbie S.; Gill, S K; Hitchcock, Edward; Beevers, D. G.

doi:10.1016/0002-9343(91)80090-9

Cited by 96 publications

(34 citation statements)

References 38 publications

Supporting

Mentioning

Contrasting

Unclassified

Order By: Relevance

“…Not all seizures in patients experiencing alcohol withdrawal are alcohol withdrawal seizures. Approximately 50% of these seizures are a result of some other organic cause, such as repetitive brain trauma; thus, alcohol withdrawal seizure is still a diagnosis of exclusion (8,32). Other signs of alcohol withdrawal, namely, autonomic hyperactivity, may not be associated with these seizures, and in chronically alcoholdependent patients, the seizures may occur while the patient has blood alcohol levels that exceed the legal limit of intoxication (33)(34)(35).…”

Section: Diagnosis and Clinical Manifestationsmentioning

confidence: 99%

Alcohol withdrawal syndromes in the intensive care unit

Sarff

Gold

2010

Critical Care Medicine

105

View full text Add to dashboard Cite

This article reviews the pathophysiology, diagnosis, and treatment of alcohol withdrawal syndromes in the intensive care unit as well as the literature on the optimal pharmacologic strategies for treatment of alcohol withdrawal syndromes in the critically ill. Treatment of alcohol withdrawal in the intensive care unit mirrors that of the general acute care wards and detoxification centers. In addition to adequate supportive care, benzodiazepines administered in a symptom-triggered fashion, guided by the Clinical Institute Withdrawal Assessment of Alcohol scale, revised (CIWA-Ar), still seem to be the optimal strategy in the intensive care unit. In cases of benzodiazepine resistance, numerous options are available, including high individual doses of benzodiazepines, barbiturates, and propofol. Intensivists should be familiar with the diagnosis and treatment strategies for alcohol withdrawal syndromes in the intensive care unit.

show abstract

Section: Diagnosis and Clinical Manifestationsmentioning

confidence: 99%

Alcohol withdrawal syndromes in the intensive care unit

Sarff

Gold

2010

Critical Care Medicine

105

View full text Add to dashboard Cite

show abstract

“…Excessive alcohol intake, especially binge drinking, has been associated with stroke [1][2][3][4][5], cardiac arrhythmias [6][7][8], sudden cardiac death [9,10], and cardiovascular mortality [11]. Furthermore, several studies have shown that excessive alcohol consumption is linked to higher prevalence of both hypertension [12][13][14][15] and target organ damage [16,17].…”

Section: Introductionmentioning

confidence: 99%

Potentiated sympathetic and hemodynamic responses to alcohol in hypertensive vs. normotensive individuals

Hering¹,

Kucharska²,

Kára³

et al. 2011

Journal of Hypertension

View full text Add to dashboard Cite

Pressor responses to acute alcohol consumption are potentiated in hypertensive patients compared with normotensive controls. Vasoconstrictor sympathetic tone is not suppressed in hypertensive patients after alcohol, despite the enhanced pressor response. Sympathetic neural mechanisms might contribute to both alcohol-related blood pressure increases and cardiovascular events in hypertensive patients.

show abstract

“…Die Hypothese einer protektiven Wirkung von Alkoholkonsum auf das Schlaganfallrisiko wird durch eine Reihe weiterer prospektiver Kohortenstudien [8,59,60,68,113] und Fallkontrollstudien [31,71,79,85] unterstützt, die im Einzelnen hier nicht weiter dargestellt werden sollen (Übersicht bei [13]). In Tabelle 2 sind die Eckdaten der größeren prospektiven Kohortenstudien zusammengefasst.…”

Section: Schützt Alkoholkonsum Vor Schlaganfall?unclassified

Alkohol und Schlaganfall Wirkt gering-mäßiger Konsum protektiv?

Schwarz

Wetterling

2002

Nervenarzt

View full text Add to dashboard Cite

It is now increasingly recognized that mild to moderate consumption of alcohol is associated with reduced mortality and coronary heart disease. Alcohol consumption and mortality follows a J-shaped association, with increasing mortality in heavy drinkers. There is some evidence from epidemiological studies that mild to moderate alcohol consumption may also reduce the risk of ischemic stroke and overall stroke in persons over 40 years of age. However, these studies have been criticized for not adequately considering possible confounding factors. Moreover, alcohol consumption is associated with an increased risk of intracerebral and sub-arachnoid hemorrhage, and binge drinking is a strong risk factor for both ischemic and hemorrhagic stroke. Alcohol consumption probably has no beneficial effects in young subjects. On the basis of the current evidence, it is not justified to advise an otherwise healthy, mild to moderate consumer of alcoholic beverages to quit drinking. However, it seems premature to advise alcohol consumption generally for prevention of stroke.

show abstract

Alcohol consumption—A risk factor for hemorrhagic and non-hemorrhagic stroke

Abstract: The results suggest that low levels of alcohol consumption may have some protective effect upon the cerebral vasculature, whereas heavy consumption predisposes to both hemorrhagic and non-hemorrhagic stroke.

Cited by 96 publications

References 38 publications

Alcohol withdrawal syndromes in the intensive care unit

Alcohol withdrawal syndromes in the intensive care unit

Potentiated sympathetic and hemodynamic responses to alcohol in hypertensive vs. normotensive individuals

Alkohol und Schlaganfall Wirkt gering-mäßiger Konsum protektiv?

Contact Info

Product

Resources

About