“…It has also been reported to complicate talc pneumoconiosis [Kleinfeld et al, 1963[Kleinfeld et al, , 1967. Evidence of cor pulmonale was noted on postmortem examination of coal miners [Rasmussen et al, 1968;Naeye and Dellinger, 1970;Naeye and Laquer, 1970;Naeye, 1971;Naeye and Dellinger, 19721, in pathological descriptions of silicotic men [Gross, 19631, and following inhalation studies in rabbits [Schepers, 19591. Recent texts on occupational lung disease include cor pulmonale among the classic complications or end-stage outcomes of the pneumoconioses [Morgan and Seaton, 1984;Parkes, 1982;Merchant, 1986;Rom, 19831, and pneumoconiosis is included among the pathogenetic mechanisms for chronic pulmonary hypertension and cor pulmonale [Fishman, 19871. However, to put this in perspective, Morgan [1984] has pointed out that findings such as those of Rasmussen et al [1968] have been dismissed [Stanescu, 19691, and emphasized that it is generally accepted that simple coal workers' pneumoconiosis seldom leads to right ventricular hypertrophy.…”