Agglutination Kinetics

Lopez, M.; Habibi, B.; Lemeud, J; Salmon, Ch.

doi:10.1111/j.1423-0410.1975.tb02741.x

Cited by 12 publications

(1 citation statement)

References 9 publications

(2 reference statements)

Supporting

Mentioning

Contrasting

Order By: Relevance

“…exhibited no symptom of any hematologic disease. Whether Rh anomaly in this patient is a precursor symptôme of a still latent disorder or it is a constitutional state without any pathologic significance may only be elucidated by a long-term hematologic observation over the following [10][11][12][13][14][15] years. It would also be interesting to investigate if Rh mosaicism is limited to red cells or if leukocytes and platelets are involved as well.…”

Section: Discussionmentioning

confidence: 99%

Two New Cases of Rh Mosaicism

Habibi

Lopez

Salmon³

1974

Vox Sanguinis

Self Cite

View full text Add to dashboard Cite

Two patients with Rh mosaicism were studied, one of whom had a myeloproliferative syndrome. The red cell mixture was found to be composed of 30% DCe/dce and 70% dce/dce populations. The separated cell components did not differ in any other marker system and their in vivo survival as well as their glycolytic enzyme content were identical. Rh quantitative and kinetic assays did not provide any evidence for monosomy in D(-) population. The data are discussed with reference to published cases of Rh mosaicism.

show abstract

Section: Discussionmentioning

confidence: 99%

Two New Cases of Rh Mosaicism

Habibi

Lopez

Salmon³

1974

Vox Sanguinis

Self Cite

View full text Add to dashboard Cite

show abstract

An auto-anti-B in an A1B person. Serological studies

Finke¹,

Sachs²,

Vollert³

et al. 1976

Blut

View full text Add to dashboard Cite

The serum of a patient (Mr. Lat) with the regular blood group A1 B contains an anti-B reacting with all cells having a B antigen except Bx and cis AB. The anti-B reacts at 4 degrees C and occasionally at room temperature as shown by agglutination, absorption-eluction and by thermo-dynamic assays. The antibody is regarded as an irregular autoantibody belonging to the group of the so called "suppressed" or "latent" antibodies.

show abstract

Ungleiches Crossing-over als mögliche Ursache blutgruppenserologischer Phänomene

Henke

Basler

1983

Z Rechtsmed

View full text Add to dashboard Cite

Unusual Serogenetic Observations Possibly Due to Unequal Crossing-overSummary. The mechanism of unequal chromosomal crossing-over is explained. Serologic pitfalls, which might be due to this abnormal genetic event, are demonstrated in the blood group systems AB0, HLA, Gin, and Rhesus.Zusammenfassung. Das Prinzip des ungleichen Crossing-overs wird erl/iutert. Ungew6hnliche Blutgruppenbefunde in den Systemen AB0, HLA, G m u n d Rhesus, die sich durch diese genetische Besonderheit erkl/iren lassen, weisen auf serene Fehlinterpretationsm6glichkeiten bei der Abstammungsbegutachtung hin.Schliisselw~Jrter: Ungleiches Crossing-over -Blutgruppen, ungleiches Crossing-over

show abstract

Agglutination Kinetics

Cited by 12 publications

References 9 publications

Two New Cases of Rh Mosaicism

Two New Cases of Rh Mosaicism

An auto-anti-B in an A1B person. Serological studies

Ungleiches Crossing-over als mögliche Ursache blutgruppenserologischer Phänomene

Contact Info

Product

Resources

About