Age dependence of fusion of primary occipital sutures: a radiographic study

Nakahara, Kuniaki; Utsuki, Satoshi; Shimizu, Satoru; Iida, Hideo; Miyasaka, Yoshio; Oka, Hiroyuki; Fujii, Kiyotaka

doi:10.1007/s00381-006-0210-8

Cited by 30 publications

(21 citation statements)

References 7 publications

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“…The occipital and innominate sutures are no longer apparent by age 4 while the mendosal suture completely fuses by 6 years of age [10]. An example of an accessory suture that can be misleading is the normal persistent occipital suture.…”

Section: Radiographic Differentiation Of Skull Fracture and Accessorymentioning

confidence: 99%

Skull fracture vs. accessory sutures: how can we tell the difference?

et al. 2010

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Section: Radiographic Differentiation Of Skull Fracture and Accessorymentioning

confidence: 99%

Skull fracture vs. accessory sutures: how can we tell the difference?

et al. 2010

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“…The latter feature appears to be found only in SGS. The ossification process may make discernment of this finding more difficult later; in normal children, the synchondrosis has closed by age 2–3 years [Nakahara et al, 2006]. The long bones often manifest broad, sclerotic appearing cortices with an appearance of inadequate tubulation of the marrow space.…”

Section: Literature Reviewmentioning

confidence: 99%

Schinzel–Giedion syndrome: Report of splenopancreatic fusion and proposed diagnostic criteria

Lehman

McFadden

Pugash

et al. 2008

American J of Med Genetics Pt A

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We report on the 46th patient with Schinzel-Giedion syndrome (SGS) and the first observation of splenopancreatic fusion in this syndrome. In the antenatal period, a male fetus was found to have bilateral hydronephrosis. Postnatally, in keeping with a diagnosis of SGS, there were large fontanelles, ocular hypertelorism, a wide, broad forehead, midface retraction, a short, upturned nose, macroglossia, and a short neck. Other anomalies included cardiac defects, widened and dense long bone cortices, cerebral ventriculomegaly, and abnormal fundi. Splenopancreatic fusion, usually encountered in trisomy 13, was found on autopsy. Schinzel-Giedion syndrome is likely a monogenic condition for which neither the heritability pattern nor pathogenesis has yet been determined. A clinical diagnosis may be made by identifying the facial phenotype, including prominent forehead, midface retraction, and short, upturned nose, plus one of either of the two other major distinguishing features: typical skeletal abnormalities or hydronephrosis. Typical skeletal anomalies include a sclerotic skull base, wide supraoccipital-exoccipital synchondrosis, increased cortical density or thickness, and broad ribs. Other highly supportive features include neuroepithelial tumors (found in 17%), hypertrichosis, and brain abnormalities. Severe developmental delay and poor survival are constant features in reported patients.

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“…Skull sutures ossify the older one gets and in some cases they become completely ossified (Nakahara et al, 2006;Cohen, 1993;Hershkovitz et al, 1997;Perizonius, 1984;Verhulst and Onghena, 1997;Pritchard et al, 1956). It was therefore decided to model the real region of cancellous bone which reduces the skull stiffness rather than a poor zigzag suture character.…”

Section: Methodsmentioning

confidence: 99%