Adhesive Capsulitis of the Hip: A Review

Looney, Colin G.; Raynor, Brett; Lowe, Rebecca

doi:10.5435/00124635-201312000-00005

Cited by 13 publications

(9 citation statements)

References 20 publications

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“…Tight anatomical connections of the inflamed synovium with the deep capsular surface produce capsular fibrosis and secondary reactive capsular thickening and retraction. 20,22 In our experience, a tension-free closure after mini posterior THR, is not possible when severe preoperative RoM restriction is present, most likely in over 50% of our patients. A tight closure of the posterior envelope after THR it is not adequate to withstand stresses generated with early mobilisation because of the long leverage arm of the lower limbs.…”

Section: Discussionmentioning

confidence: 79%

Tension-free closure with capsular lengthening in mini posterior total hip replacement

Santori

2018

HIP International

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Choice of the best mini invasive surgical approach for total hip replacement remains a controversial topic. The posterior approach is traditionally associated with a higher dislocation rate and the obligation of postoperative restrictions. Soft tissue repair reduces the risk of dislocation yet, posterior closure it is often challenging because of capsular and external rotator contractures. In this article, an original procedure of capsular lengthening and tension-free closure is described and the results of a retrospective single surgeon series of minimally invasive posterior total hip replacement (THR) with capsular repair are presented. Methods: A total of 925 mini posterior total hip replacements performed between 2009 and 2015 were retrospectively reviewed. 2 original types of capsulorrhaphy to decrease tension of the repaired tissues were employed. Absence of posterior envelope stretch during flexion and internal rotation was confirmed intraoperatively with a dynamic test. No postoperative restrictions were used. Results: Capsular lengthening with 1 of the described techniques was required in over 50% of cases for a tensionfree closure. 3 patients dislocated in the 867 reviewed procedures (0.35%), 1 requiring revision for instability (0.11%). Conclusions: This soft tissue repair technique after mini posterior approach THR is technically easy, provides a tensionfree closure and a low dislocation rate.

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Section: Discussionmentioning

confidence: 79%

Tension-free closure with capsular lengthening in mini posterior total hip replacement

Santori

2018

HIP International

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“…There is some literature about a similar condition in hips, ankles and also knees. However, the currently available literature are mainly case reports of conditions seldomly seen in clinical practice [ 108 , 109 ]. Contractures with fibrosis do occur frequently mainly in knees and elbows, but without the potential for spontaneous recovery as FS has.…”

Section: Discussionmentioning

confidence: 99%

The puzzling pathophysiology of frozen shoulders – a scoping review

et al. 2020

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Purpose The pathophysiology of frozen shoulders is a complex and multifactorial process. The purpose of this review is to scope the currently available knowledge of the pathophysiology of frozen shoulders. Methods A systematic search was conducted in Medline, Embase and the Cochrane library. Original articles published between 1994 and October 2020 with a substantial focus on the pathophysiology of frozen shoulders were included. Results Out of 827 records, 48 original articles were included for the qualitative synthesis of this review. Glenohumeral capsular biopsies were reported in 30 studies. Fifteen studies investigated were classified as association studies. Three studies investigated the pathophysiology in an animal studies. A state of low grade inflammation, as is associated with diabetes, cardiovascular disease and thyroid disorders, predisposes for the development of frozen shoulder. An early immune response with elevated levels of alarmins and binding to the receptor of advance glycation end products is present at the start of the cascade. Inflammatory cytokines, of which transforming growth factor-β1 has a prominent role, together with mechanical stress stimulates Fibroblast proliferation and differentiation into myofibroblasts. This leads to an imbalance of extracellular matrix turnover resulting in a stiff and thickened glenohumeral capsule with abundance of type III collagen. Conclusion This scoping review outlines the complexity of the pathophysiology of frozen shoulder. A comprehensive overview with background information on pathophysiologic mechanisms is given. Leads are provided to progress with research for clinically important prognostic markers and in search for future interventions. Level of evidence Level V.

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“…Radiographs may differentiate the 2 conditions, because no or little hip arthritis will be noted in patients with adhesive capsulitis. At the time of this review, the literature contains 13 reports of this disorder; however, some of these reports describe concomitant hip OA and deformity, bringing the diagnosis into question [41]. Hip adhesive capsulitis is diagnosed when ROM is limited in all planes of motion with the patient in a state of general anesthesia.…”

Section: Differential Diagnosis Of Hip Painmentioning

confidence: 99%

“…Conservative treatment of adhesive capsulitis is identical to the treatment of hip OA. If conservative treatment fails, examination of the hip with the patient in a state of general anesthesia is performed and arthroscopic versus open capsular release can be performed [41].…”

Section: Treatments For Common Local Causes Of Hip Girdle Painmentioning

confidence: 99%

Diagnosis and Treatment of Hip Girdle Pain in the Athlete

Prather

Cheng

2016

PM&R

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Evaluation of an athlete's report of "hip pain" is challenging. Many conditions involving the pelvic girdle can present with overlapping pain distributions, and athletes often may have coexisting disorders. Appropriate evaluation requires thorough, systematic consideration of intra-articular hip disease, extra-articular local causes of hip pain, and referred pain from other musculoskeletal or even visceral sources. Although our understanding of intra-articular hip disorders has been greatly enhanced in recent decades through advances in hip arthroscopy, gaps still exist in our understanding of appropriate treatment, including effective nonsurgical protocols and when to consider surgical intervention. For instance, we have a better understanding of hip dysfunction related to structural changes that occur prior to the onset of arthritis, but we are also learning that correction of the structural changes does not always guarantee a correction of dysfunction and pain. Furthermore, athletes need instruction and guidance in retraining appropriate movement patterns after a surgical intervention. Risk stratification regarding which athletes need surgical intervention and what their needs are after specific surgical procedures remain undefined. In this review we will describe the differential diagnosis of hip and pelvic girdle pain in the athlete and then discuss how to use a history, physical examination, and appropriate imaging and diagnostic injections to arrive at a proper diagnosis. Lastly, a brief discussion is included of key concepts and controversies involved in treating some of the most common hip disorders experienced by athletes.

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Adhesive Capsulitis of the Hip: A Review

Cited by 13 publications

References 20 publications

Tension-free closure with capsular lengthening in mini posterior total hip replacement

Tension-free closure with capsular lengthening in mini posterior total hip replacement

The puzzling pathophysiology of frozen shoulders – a scoping review

Diagnosis and Treatment of Hip Girdle Pain in the Athlete

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