Acute thrombosis of the sinus node artery: arrhythmological implications

Andò, Giuseppe; Gaspardone, Achille; Proietti, Igino

doi:10.1136/heart.89.2.e5

Cited by 34 publications

(23 citation statements)

References 9 publications

(7 reference statements)

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“…3 Ischemia of the SN artery is generally considered to be an uncommon pathogenic mechanism of SN dysfunction, however, cases of SN dysfunction due to ischemia have occasionally been reported. 4 We present the case of an occurrence of an AF episode as an ischemic complication in a patient with an SN artery originating from the left circumflex artery (LCx) undergoing stenting of the LCx.…”

Section: Introductionmentioning

confidence: 99%

Subocclusion of the Sinus Node Artery During Coronary Angioplasty: Arrhythmological Considerations

D’Ascenzi

Iadanza

Zacà

et al. 2010

Clinical Cardiology

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Section: Introductionmentioning

confidence: 99%

Subocclusion of the Sinus Node Artery During Coronary Angioplasty: Arrhythmological Considerations

D’Ascenzi

Iadanza

Zacà

et al. 2010

Clinical Cardiology

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“…4,10 -12 However, the collateral circulation is often insufficient, and sinus node ischemia due to coronary artery occlusion, proximal to the origin of the sinus node artery, can result in atrial bradyarrhythmias or tachyarrhythmias, especially atrial fibrillation. 10,[12][13][14] Therefore, in many individuals, atherosclerosis and compromise of the left circumflex coronary artery can result in significant morbidity and death via the effects on the atria alone.…”

Section: Discussionmentioning

confidence: 99%

Left Atrial Infarction

Rose

Collins²

2010

American Journal of Forensic Medicine &Amp; Pathology

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The majority of cardiac related deaths are due to ischemic heart disease, with the most common clinical scenario being severe coronary artery atherosclerosis resulting in left ventricular myocardial infarction. However, infarction of other cardiac chambers does occur, and often has specific clinical associations. We report a case of a 70-year-old man who suffered from left atrial infarction that resulted in a transmural rupture of his left atrium. The patient had a history of rheumatic heart disease, mitral valve stenosis, and severe atherosclerotic coronary artery disease. Four days before death, he underwent mitral valve replacement and left circumflex coronary artery bypass. Two days later, he developed atrial fibrillation. On the day of death, he had decreased mental status, questionable seizure activity, hematemesis, ventricular tachycardia, and eventually asystole. At autopsy, he had significant hemopericardium with a fibrinous pericarditis and bilateral hemothoraces (total blood volume: 1250 mL). A 0.1 to 0.2 cm left atrial transmural defect was identified. The prosthetic mitral valve was free of vegetations, and completely intact. Similarly, the left circumflex artery bypass graft was completely patent and unremarkable. Severe calcific atherosclerosis was of his native left circumflex and left main coronary arteries. Microscopic examination revealed acute myocardial infarction of the left atrium at the rupture site. The anatomy of atrial circulation as well as the pathology and consequences of atrial infarction are discussed.

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“…Indeed, not infrequently, the ischemic hypothesis of AF usually is not taken into great consideration, even if it could be an epiphenomenon appearing during acute hypoxia, pneumonia, pulmonary embolism . Finally, it should be said that even the acute thrombosis of the SN artery has been implied into sinus node dysfunction, in the context of coronary artery disease …”

Section: Discussionmentioning

confidence: 99%