Acute risk factors for myocardial infarction

Triggering of supraventricular premature beats. The impact of acute and chronic risk factors

“…Emotional stress, a well-documented trigger of ventricular arrhythmias [8][9][10]12] and acute coronary syndrome [20][21][22], showed no association with the frequency of SPB in the present study. A previous myocardial infarction was associated with the lower occurrence of SPB in those under the 65 years of age which may be associated with autonomic abnormalities following myocardial infarction [23,24].…”

The Emotional, Environmental, Physical and Chemical Triggers of Acute Myocardial Infarction: An Analytical Demographic Study

“…The cold seasons causes physiological changes including increases in blood sugar, levels of cholesterol, fibrinogen concentration and platelet aggregation. Fibrinogen plays an important role in the formation of clots in the coronary artery, the start of acute myocardial infarction and life-threatening arrhythmias (7). Exposure to high temperature could increase the viscosity of plasma and cholesterol levels in serum (17).…”

“…Some of these triggers like physical activities are present in half of the initiation cases of acute coronary syndromes in men. Acute triggers include emotional, physical, environmental and chemical triggers (7)(8)(9). Triggers lead into the start of hemodynamic, prothrombotic, and vascular constrictive procedures which are accompanied by an increase in the risk of cardiovascular diseases and all occur in some hours (7,10).…”

“…The presence of plaques susceptible to atherosclerosis, disturbance in the conductive system of the heart, and small arteries diseases along with physical and emotional trigger stressors, could transiently cause vasoconstriction and prothrombic effects which all finally lead into plaque rupture and thrombosis. The trigger can cause ventricular fibrillation and sudden death by decreasing the cardiac electrical resistance threshold and increasing sympathetic activity through releasing central mediators such as catecholamine (7,13).…”

Acute myocardial infarction in a 24 year-old man possibly associated with sibutramine use

“…Several reports documented that elevations in plasma adrenaline and noradrenaline levels may increase the risk of acute coronary syndromes. High noradrenaline levels not only increase blood pressure and pulse rate, also directly promote a prothrombotic and vasoconstrictive state which may all cause instability of an existing atherosclerotic plaque that would result in rupture [8][9][10]. Such 'sympathetic triggering' is a recognized underlying mechanism for AMI particularly at early morning hours and in patients without previous anginal complaints [9,10].…”