Acute Right Ventricular Dysfunction in a Critically Ill Patient with COVID-19

Venkata, Chakradhar; Aruchamy, Senthil; Kasal, Jan

doi:10.1016/j.case.2020.08.007

Cited by 2 publications

(11 citation statements)

References 22 publications

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“…Veronese et al 31 described fulminant myocarditis triggered by OC43 subtype coronavirus in an Italian patient. The study from the United States of America by Venkata et al 32 registered a case of acute right ventricular dysfunction in a critically ill 67-yearold patient with COVID-19. Dabbagh et al 33 registered a cardiac tamponade secondary to COVID-19 in a 67-year-old man from the United States of America.…”

Section: Results Of Individual Studiesmentioning

confidence: 99%

“…Regarding the comorbidities associated with the clinical status of the patients, the most prevalent were systemic arterial hypertension (27.3%) 23,28,36,38,41,43 and type 2 diabetes mellitus (22.7%). 32,36,38,39,41 However, it is worth noting that 31.8% of the patients did not present comorbidities, 23,26,27,34,35,40,44 as shown in Table 2.…”

Section: Comorbiditiesmentioning

confidence: 94%

“…The ECG showed that most patients presented elevation of the ST segment (31.8%). 25,34,36,38,40,41,44 In addition, six studies (27.3%) reported normality in their patients, 24,30,32,35,37,42 as shown in Table 3.…”

Section: Ecg Changesmentioning

confidence: 98%

“…Most patients had fever at hospital admission, accounting for 54.5% of the articles that reported myocarditis in COVID-19. [25][26][27]31,34,[36][37][38][40][41][42][43] Subsequently, symptoms such as dyspnea (54.5%), 26 27,30-33,35-37,39,40,43 cough (45.4%), 26,27,29,[32][33][34][38][39][40][41] and chest pain (22.7%), 24,25,28,29,35 respectively, affected patients (Table 2).…”

Section: Clinical Manifestationsmentioning

confidence: 99%

“…The increased levels of troponin I or troponin T (63.6%) 24,25,[28][29][30][31][32][33][34][36][37][38][42][43][44] were the most recurrent hemodynamic alterations in patients who developed cardiac involvement due to COVID-19. There were frequent reports of oxygen saturation (SaO 2 ) below 95% (40.9%), 24,26,31,34,35,37,38,41,43 increased D-dimer (31.8%), 27,29,32,36,37,41,42 evolution of patients to cardiogenic shock (22.7%), 27,31,36,38,44 and occurrence of metabolic acidosis (18.1%). 27,31,37,44…”

Section: Hemodynamic Aspectsmentioning

confidence: 99%

See 4 more Smart Citations

Covid-19 and Heart Involvement: A Systematic Review of Literature

Moraes¹,

Santos²,

Campos³

et al. 2023

International Journal of Cardiovascular Sciences

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Background: Recent reports in the literature have indicated that infection by coronavirus disease 2019 (COVID-19) causes cardiac complications, such as heart failure, arrhythmia, myocardial infarction, and even fulminant myocarditis. These complications have been identified as the cause of death in some patients infected with SARS-CoV-2. Objectives:To analyze echocardiographic and electrocardiographic changes, treatments used, and clinical outcomes in patients with myocarditis and COVID-19. Methods:The items described for Preferred Reporting Items for Systematic Reviews and Meta-Analyses (PRISMA) were followed. This review included articles in English, Portuguese, and Spanish that reported cardiac involvement, injury, or myocardial inflammation in patients who acquired COVID-19 (SARS-CoV-2).Results: Five databases were consulted to find 1,726 articles. After applying the eligibility criteria, a total of 22 studies were considered qualified. ST-segment (section of the electrocardiogram corresponding the end of the S wave to the beginning of the T wave) elevation and tachyarrhythmia were the most common changes found in the electrocardiographic analysis of patients affected with COVID-19. Concerning echocardiography, there was a high frequency of decreased ejection fraction and occurrence of pericardial effusion. Conclusion:This systematic review provides a potential tool for the analysis of cardiac changes and implications caused in patients affected by SARS-CoV-2 infection, with emphasis on the presence of tachyarrhythmia on electrocardiogram (ECG) and decreased ejection fraction on echocardiogram.

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Section: Results Of Individual Studiesmentioning

confidence: 99%

Section: Comorbiditiesmentioning

confidence: 94%

Section: Ecg Changesmentioning

confidence: 98%

Section: Clinical Manifestationsmentioning

confidence: 99%

Section: Hemodynamic Aspectsmentioning

confidence: 99%

See 3 more Smart Citations

Covid-19 and Heart Involvement: A Systematic Review of Literature

Moraes¹,

Santos²,

Campos³

et al. 2023

International Journal of Cardiovascular Sciences

View full text Add to dashboard Cite

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Understanding COVID-19-associated coagulopathy: From PIC to SIC or DIC

Wang¹,

Gao²,

He³

et al. 2021

Journal of Intensive Medicine

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Coagulopathy, characterized by a high D-dimer level, is a common pathological occurrence in coronavirus disease 2019 (COVID-19) and is associated with poor prognosis. Severe cases with COVID-19 is associated with a significantly higher risk of deep vein thrombosis and acute pulmonary embolism. Pulmonary intravascular coagulopathy is the characteristic coagulopathy in COVID-19. Unlike sepsis-induced coagulopathy and disseminated intravascular coagulation, which are manifestations of systemic coagulopathy, pulmonary intravascular coagulopathy is a manifestation of a local coagulation disorder in the lung. The progression from pulmonary intravascular coagulopathy to sepsis-induced coagulopathy or disseminated intravascular coagulation in the context of COVID-19 may indicate that the patient's coagulation dysfunction has progressed from local to systemic. Exploring the associated coagulation disease will aid in the understanding of the pathophysiological mechanisms underlying severe COVID-19.

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Acute Right Ventricular Dysfunction in a Critically Ill Patient with COVID-19

Cited by 2 publications

References 22 publications

Covid-19 and Heart Involvement: A Systematic Review of Literature

Covid-19 and Heart Involvement: A Systematic Review of Literature

Understanding COVID-19-associated coagulopathy: From PIC to SIC or DIC

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