“…It has been suggested that CBS risk is related more with the dynamic decrease of vision in sight-impaired patients or the acute decline of vision, rather than the low visual acuity itself (4). One of the most prevalent theories suggests that the acute visual impairment is a result of the hyperactivation of visual-related cortex, due to changes in neurotransmission during the early recovery period (1,3,5). The above theory could explain our case, in which the recovery period was short, with only 3 days of symptoms, and the cessation of the visual hallucinations indicated the end of it.…”