Acute Pulmonary Edema and Cardiac Failure Due to Zinc Phosphide Ingestion

Söğüt, Özgür; Baysal, Zeynep; Özdemir, Bora

doi:10.1016/j.jemermed.2009.05.039

Cited by 29 publications

(37 citation statements)

References 4 publications

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“…Also, some rare complications have been reported such as pulmonary edema 2, acute pancreatitis, transient leukopenia, and transient hyperglycemia 4, 5, 6.…”

Section: Introductionmentioning

confidence: 99%

Hepatotoxicity due to zinc phosphide poisoning in two patients: role of N‐acetylcysteine

Oghabian

Afshar

Rahimi

2016

Clinical Case Reports

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Key Clinical MessageZinc phosphide (Zn3P2/ZnP) is used as a rodenticide. The most common signs of toxicity are nausea, vomiting, hypotension, and metabolic acidosis; patients presenting such signs are referred to the emergency department (ED) of the hospitals. Therefore, this study aimed to report two cases of hepatotoxicity following accidental and intentional ZnP poisoning and successful management with N‐acetylcysteine (NAC).

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“…Also, some rare complications have been reported such as pulmonary edema 2, acute pancreatitis, transient leukopenia, and transient hyperglycemia 4, 5, 6.…”

Section: Introductionmentioning

confidence: 99%

Hepatotoxicity due to zinc phosphide poisoning in two patients: role of N‐acetylcysteine

Oghabian

Afshar

Rahimi

2016

Clinical Case Reports

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show abstract

“…Profuse vomiting, abdominal pain, palpitation, dyspnoea and tachypnea are the most common presenting features. Phosphide causes circulatory failure resulting in congestion and oedema of most organs particularly, in the lungs [6]. Other features include disseminated intravascular coagulation, metabolic acidosis, or mixed metabolic acidosis and respiratory alkalosis, acute renal failure and ultimately multi-organ failure.…”

Section: Discussionmentioning

confidence: 99%

A case of severe zinc phosphide poisoning

Gunaratne¹,

Wijeratne²,

Pilapitiya³

et al. 2016

Ceylon Med. J.

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“…70.8% of those with depressed sensorium died, with refractory state of shock and encephalopathy due to hepatic failure being the cause of high mortality. It has been proven that the systemic toxicity caused by phosphine is mainly to the heart, lungs, liver, gastrointestinal tract, kidney, and brain since PH 3 molecules targets them particularly (23,24). The effect of phosphine causing failure of the heart, lungs, and gastrointestinal tract is well studied in cases of aluminum phosphide poisoning, but the effect of it in the liver and renal parenchyma is not well studied (12,19,21).…”

Section: Discussionmentioning

confidence: 99%