2001
DOI: 10.1046/j.1365-2141.2001.03167.x
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Acute event‐related anaemia

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Cited by 18 publications
(7 citation statements)
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“…No change (Faraco et al, 2015b) Increase (slight) (Faraco et al, 2015b) SvO2 Increase (Faraco et al, 2015b) Increase (Faraco et al, 2015b) * Assumed (See section 4.3) † Contributing effects of hypocapnia for non-isocapnic stimuli Hct typically ranges between 36-54% (Billett, 1990), with natural fluctuations arising as a function of age, (Adeli et al, 2015;Daniel, 1973) and potentially race (Beutler and West, 2017;Cheng et al, 2004). The need for individualised knowledge of Hct becomes critical in anaemia, with significant reductions observed in a wide range of pathology (Babitt and Lin, 2012;Goodnough et al, 2003;Muncie and Campbell, 2009;Rees et al, 2010;van Iperen et al, 2001), secondary to therapy (Groopman and Itri, 1999;Ludwig et al, 2004;Rodgers et al, 2012), and in response to poor nutrition (Baker and Demaeyer, 1979 (Joseph et al, 2016;Li et al, 2017). Furthermore, Hct is known to be reduced in microvasculature in comparison to larger vessels.…”
Section: Sao2mentioning
confidence: 99%
“…No change (Faraco et al, 2015b) Increase (slight) (Faraco et al, 2015b) SvO2 Increase (Faraco et al, 2015b) Increase (Faraco et al, 2015b) * Assumed (See section 4.3) † Contributing effects of hypocapnia for non-isocapnic stimuli Hct typically ranges between 36-54% (Billett, 1990), with natural fluctuations arising as a function of age, (Adeli et al, 2015;Daniel, 1973) and potentially race (Beutler and West, 2017;Cheng et al, 2004). The need for individualised knowledge of Hct becomes critical in anaemia, with significant reductions observed in a wide range of pathology (Babitt and Lin, 2012;Goodnough et al, 2003;Muncie and Campbell, 2009;Rees et al, 2010;van Iperen et al, 2001), secondary to therapy (Groopman and Itri, 1999;Ludwig et al, 2004;Rodgers et al, 2012), and in response to poor nutrition (Baker and Demaeyer, 1979 (Joseph et al, 2016;Li et al, 2017). Furthermore, Hct is known to be reduced in microvasculature in comparison to larger vessels.…”
Section: Sao2mentioning
confidence: 99%
“…За поширеністю, АХЗ є другою після ЗДА у хворих із гострими або хронічними імуноактивними процесами, з чим і пов`язана її друга назва «анемія хронічного запалення» [7][8][9][10]. Найчастіше така анемія асоційована із інфекційними -гострими та хронічними бактеріальними, паразитарними та грибковими захворюваннями (18-95% випадків) [11][12][13], онкологічними процесами -30-77% [12][13][14][15][16], автоімунними (ревматоїдний артрит, васкуліти, саркоїдоз, запальні захворювання товстої кишки) -8-71% [17][18][19][20], хронічними захворюваннями нирок -23-50% [21][22][23]. Зазвичай, АХЗ є нормоцитарною та нормохромною [8][9][10]24], але АХЗ може бути гіпопроліферативною, а іноді й мікроцитарною, тому диференціальна діагностика має включати залізодефіцитну анемію (ЗДА).…”
Section: Indicators Of Iron Exchanges In Patients With Chronic Obstruunclassified
“…An acute variant of ACD has been reported in patients with major trauma, myocardial infarction, and sepsis [300,307] . …”
Section: Anemia Of Chronic Diseasementioning
confidence: 99%