Acute cilio-choroidal effusion due to acetazolamide: unusual posterior involvement (OCT aspects)

Malagola, R; Giannotti, Rossella; Pattavina, Luigi; Arrico, L.

doi:10.1038/eye.2013.41

Cited by 12 publications

(12 citation statements)

References 5 publications

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“…In addition, we advise caution on treating patients with systemic carbonic anhydrase inhibitors, as there have been reports of acetazolamide causing an idiosyncratic reaction characterized by transient myopia, ciliary body edema, uveal effusions, anterior rotation of the lens-iris diaphragm, and acute angle-closure glaucoma. [14][15][16][17] Management in refractory cases with argon laser iridoplasty has been reported. 18 Rhee et al 19 first reported the successful use of mannitol and intravenous methylprednisone to treat patients with this condition, and suggested that the response to steroids supported the theory that ciliochoroidal effusion is secondary to an inflammatory etiology.…”

Section: Discussionmentioning

confidence: 97%

Bilateral Angle Closure Following Use of a Weight Loss Combination Agent Containing Topiramate

Grewal

Goldstein

Khatana

et al. 2015

Journal of Glaucoma

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Section: Discussionmentioning

confidence: 97%

Bilateral Angle Closure Following Use of a Weight Loss Combination Agent Containing Topiramate

Grewal

Goldstein

Khatana

et al. 2015

Journal of Glaucoma

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“…Early case reports about bilateral choroidal effusion and acute angle closure were most commonly associated with the use of topiramate [2] . There have been a few bilateral cases reported after acetazolamide following monocular cataract surgery [3] , [5] , [6] , [7] , [8] . We present a case of bilateral ciliochoroidal effusion syndrome after exposure to acetazolamide in a subject with plateau iris configuration, normal lens, and without any surgery.…”

Section: Introductionmentioning

confidence: 99%

Acetazolamide-induced bilateral ciliochoroidal effusion syndrome in plateau iris configuration

Man

Costa

Ayres

et al. 2016

American Journal of Ophthalmology Case Reports

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Purpose Our purpose is to describe a 60-year-old male, who has plateau iris configuration and developed bilateral ciliochoroidal effusion syndrome after ingestion of acetazolamide. Observations Our case was a research participant in a multi-center clinical study (ClinicalTrials.gov NCT01677507). During the course of this study, he was treated with a single dose of acetazolamide (500 mg), and seven days later treated with latanoprost one drop daily at bedtime both eyes for seven days, and then was administered another dose of acetazolamide (500 mg). Several hours later he complained of blurred vision in the distance and mild headache. On examination, he had a myopic shift, intraocular pressures of 36 mmHg in right eye and 35 mmHg in left eye, shallow anterior chambers both eyes, and occluded angles by gonioscopy both eyes. An echographic exam confirmed the bilateral ciliochoroidal effusion syndrome. He was treated by no further dosing of acetazolamide and started on timolol, atropine and prednisolone. Two weeks later, the bilateral choroidal effusion and acute angle closure were resolved. Repeat echography showed plateau iris configuration. Conclusions and Importance To the best of our knowledge, drug-induced bilateral ciliochoroidal effusion syndrome has not been reported with acetazolamide in plateau iris configuration.

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“…Some authors stated that preservation of photoreceptors after surgery may be due to the diffusion of cytokines and growth factors originated from the stimulated ciliar body, iris, and retina [19–22]. They are basic fibroblast growth factor (bFGF) [23], neurotrophic growth factor (CNTF) [24], brain-derived neurotrophic factor (BDNF) [25], nerve growth factor (NGF) [26], and lens-epithelium-derived growth factor (LEGF) [27].…”

Section: Discussionmentioning

confidence: 99%

Microincision Vitrectomy Surgery in Vitreomacular Traction Syndrome of Retinitis Pigmentosa Patients

Gerace

Valente

Spadea

et al. 2014

BioMed Research International

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Purpose. To investigate long-term retinal changes after microincision pars plana vitrectomy surgery (MIVS) and internal limiting membrane (ILM) peeling outcome in retinitis pigmentosa (RP) patients affected by vitreomacular traction syndrome (VMT) with higher vitreous surface adhesion or coexisting epiretinal membrane (ERM). Methods. Eight RP patients suffering from VMT were evaluated by means of best corrected visual acuity (BCVA), anterior and posterior binocular examination, spectral-domain optical coherence tomography (SD-OCT), MP-1 microperimetry (MP-1), and full-field electroretinogram (ERG), before MIVS and ILM peeling and during the 36-month follow-up. Patients were hospitalized for two days after the surgery. Surgical procedure was performed following this schedule: surgical removal of crystalline lens (MICS), MIVS with 23-gauge sutureless system trocars, core vitreous body removal, and balanced-sterile-salin-solution- (BSS-) air-gas (SF6) exchange. Results. All patients presented visual acuity (VA) increase after MIVS. None of the patients developed ocular hypertension or vitreomacular adhesions during the 3-year follow-up. MP-1 bivariate contour ellipse area (BCEA) was reduced in its dimensions and improved in all patients demonstrating a better fixation. Conclusions. MIVS could be the gold standard therapy in RP patients with VMT and higher vitreous surface adhesion or coexisting ERM if medical therapy is not applicable or not effective.

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Acute cilio-choroidal effusion due to acetazolamide: unusual posterior involvement (OCT aspects)

Cited by 12 publications

References 5 publications

Bilateral Angle Closure Following Use of a Weight Loss Combination Agent Containing Topiramate

Bilateral Angle Closure Following Use of a Weight Loss Combination Agent Containing Topiramate

Acetazolamide-induced bilateral ciliochoroidal effusion syndrome in plateau iris configuration

Microincision Vitrectomy Surgery in Vitreomacular Traction Syndrome of Retinitis Pigmentosa Patients

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