Acquired Pial Arteriovenous Fistula Following Cerebral Vein Thrombosis

Phatouros, Constantine C.; Halbach, Van V.; Dowd, Christopher F.; Lempert, Todd E.; Malek, Adel M.; Meyers, Philip M.; Higashida, Randall T.

doi:10.1161/01.str.30.11.2487

Cited by 68 publications

(46 citation statements)

References 27 publications

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“…MRI and MR venography or 3D-XRA DSA venography can speed up the diagnostic process in a subgroup of patients with deep veins involvement and rapid clinical symptoms progression. Thrombolytic therapy can also prevent or reduce the risk of secondary complications after CVT like chronic hih intracranial preassure with visual loss and/or secondary pial or dural arteriovenous malformations 23,24 .…”

Section: Discussionmentioning

confidence: 99%

Oral Contraceptive Induced Cerebral Venous Thrombosis Treated by Local Catheter Directed Thrombolysis

Procházka¹,

Rajner

Procházka

et al. 2004

Interv Neuroradiol

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We report on a case of cerebral venous thrombosis (CVT) induced by oral contraception (OC) activated coagulopathy and its endovascular treatment. Deep venous system and dural sinuses thrombosis complicated with severe neurological deficit and coma due to right thalamic edema and ischemia in a young woman was treated by local thrombolysis with an administration of 0.6 mg/h of the rtPA and the concomitant intravenous unfractioned heparin infusion (700 IU/h). 3D-Xra digital rotational venography performed at the beginning and after treatment confirmed thrombus resolution with rapid flow restoration. Dynamic flow imaging gives interesting information on the deep venous system and the cortical venous collectors drainage. Final NIHSS (National Institute of Health Stroke Scale) and mRS (modified Rankin Scale) confirmed an excellent clinical outcome of the interventional therapy.

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Section: Discussionmentioning

confidence: 99%

Oral Contraceptive Induced Cerebral Venous Thrombosis Treated by Local Catheter Directed Thrombolysis

Procházka¹,

Rajner

Procházka

et al. 2004

Interv Neuroradiol

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“…236 A pial fistula can also follow a cortical vein thrombosis. The relationship between the 2 entities is rather complex, because (1) dural fistulas can be a late complication of persistent dural sinus occlusion with increased venous pressure, (2) the fistula can close and cure if the sinus recanalizes, and (3) a preexisting fistula can be the underlying cause of CVT.…”

Section: Dural Arteriovenous Fistulamentioning

confidence: 99%

“…72,179,201,236,263 In the Canadian Pediatric Ischemic Stroke Registry, 85 of 160 children with CVT at 16 Canadian children's hospitals received anticoagulation (25 neonates and 60 non-neonates). There were no fatal or severe complications reported; however, follow-up was not systematic.…”

Section: Primary Evidencementioning

confidence: 99%

Diagnosis and Management of Cerebral Venous Thrombosis

Saposnik¹,

Barinagarrementería²,

Brown³

et al. 2011

Stroke

1,638

1,229

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Background— The purpose of this statement is to provide an overview of cerebral venous sinus thrombosis and to provide recommendations for its diagnosis, management, and treatment. The intended audience is physicians and other healthcare providers who are responsible for the diagnosis and management of patients with cerebral venous sinus thrombosis. Methods and Results— Members of the panel were appointed by the American Heart Association Stroke Council's Scientific Statement Oversight Committee and represent different areas of expertise. The panel reviewed the relevant literature with an emphasis on reports published since 1966 and used the American Heart Association levels-of-evidence grading algorithm to rate the evidence and to make recommendations. After approval of the statement by the panel, it underwent peer review and approval by the American Heart Association Science Advisory and Coordinating Committee. Conclusions— Evidence-based recommendations are provided for the diagnosis, management, and prevention of recurrence of cerebral venous thrombosis. Recommendations on the evaluation and management of cerebral venous thrombosis during pregnancy and in the pediatric population are provided. Considerations for the management of clinical complications (seizures, hydrocephalus, intracranial hypertension, and neurological deterioration) are also summarized. An algorithm for diagnosis and management of patients with cerebral venous sinus thrombosis is described.

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“…11) Likewise, the pathophysiological mechanisms underlying acquired pial AVF remain to be elucidated, but, like dural AVF, development has been attributed to venous hypertension following head injury, brain surgery, and to venous thrombosis. 11,16,18) Our patient was an adult female without Rendu-Osler-Weber or Klippel-Trenaunay-Weber disease. She had no obvious history of head trauma or brain surgery, but transient venous hypertension following slight trauma or of asymptomatic venous thrombosis with recanalization cannot be excluded completely.…”

Section: Discussionmentioning

confidence: 94%

Intracranial Pial Arteriovenous Fistula -Case Report-

Yamashita

Ohe

Yoshimura

et al. 2007

Neurol. Med. Chir.(Tokyo)

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A 33-year-old woman presented with a rare intracranial pial arteriovenous fistula manifesting as monoparesis and hypesthesia of the right lower extremity. Computed tomography demonstrated an approximately 10-mm diameter subcortical hematoma in the left postcentral gyrus. Two months after suffering the ictus, angiography demonstrated a pial arteriovenous fistula in the late arterial phase fed by the left paracentral artery and drained into the left precentral vein. No nidus or dural arteriovenous fistula was detected. Left parietal craniotomy was performed and the pial arteriovenous fistula was extirpated by electrocoagulation. Intraoperative angiography demonstrated disappearance of the fistula. She experienced no postoperative neurological deterioration, but hypesthesia of the right leg persisted. Obliteration of the pial arteriovenous fistula was reconfirmed by postoperative angiography. She suffered no rebleeding episodes during the 36-month follow-up period. Pial arteriovenous fistula causing mild symptoms should be treated by flow disconnection because the direct arteriovenous shunt and attendant high blood flow usually results in huge venous varices. To determine whether direct surgery or endovascular treatment is appropriate, the position and shape of the lesion must be known.

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Acquired Pial Arteriovenous Fistula Following Cerebral Vein Thrombosis

Cited by 68 publications

References 27 publications

Oral Contraceptive Induced Cerebral Venous Thrombosis Treated by Local Catheter Directed Thrombolysis

Oral Contraceptive Induced Cerebral Venous Thrombosis Treated by Local Catheter Directed Thrombolysis

Diagnosis and Management of Cerebral Venous Thrombosis

Intracranial Pial Arteriovenous Fistula -Case Report-

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