Acquired deep venous thrombosis in children

Marciński, A; Barral, V.; Sauvegrain, J

doi:10.1007/bf02386762

Cited by 8 publications

(4 citation statements)

References 27 publications

(31 reference statements)

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“…Large retrospective studies of symptomatic children whose ages spanned 4-21 years found only 36 children with a confirmed DVT, the majority in the lower extremities [8][9][10][11][12]. The size of the population was given in only one study (19 cases out of 24,250 pediatric admissions) [13].…”

Section: Discussionmentioning

confidence: 99%

Prevalence of deep venous thrombosis in the lower extremities of children in the intensive care unit

et al. 1996

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Section: Discussionmentioning

confidence: 99%

Prevalence of deep venous thrombosis in the lower extremities of children in the intensive care unit

et al. 1996

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“…Thrombus formation in vivo occurs preferentially when the blood flow is disturbed and the clotting system is stimulated, for example as a result of illness (6), in response to the puncture of a vessel caused by the catheter (9), or as a reaction to artificial catheter material (19)(20)(21).…”

Section: Discussionmentioning

confidence: 99%

“…One of the most frequent complications affecting implanted catheters is that of thrombus formation (1)(2)(3). In several studies up to 20% of all patients exhibited catheter failure resulting from thrombotic occlusion of the catheter lumen (4)(5)(6).…”

Section: Introductionmentioning

confidence: 99%

Occlusive Thrombus Formation on Indwelling Catheters: In Vitro Investigation and Computational Analysis

Friedrich

Reininger²

1995

Thromb Haemost

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SummaryTo determine the hydrodynamic parameters involved in thrombus formation on indwelling catheters, fibrin coagulation was investigated in an in vitro model and compared with the prevailing flow field. Computational analysis was used to simulate clot growth and identify the variables which determine its localization. The fibrin clot grew from the distal end of the catheter along the interface between the bulk stream and the annular vortex located immediately downstream of the catheter tip, finally filling the entire vortex. The calculations of flow parameters for three different growth stages showed that regions of clot formation always correlated with regions exhibiting the longest residence time. Fibrin clotting could be simulated by a mathematical model using residence time as the only variable parameter. Our results demonstrate that the residence time is the single most important flow factor for fibrin coagulation and thus probably for thrombus formation. When the residence time equals the clotting time for the fluid, thrombus formation is initiated. Conversely, it ceases when the area of long fluid residence times is replaced by a fibrin clot. Hence the fibrin clot represents a hydrodynamic optimization of the previously disturbed flow.

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“…DVT has also been reported previously in association with other infectious agents, including Salmonella typhimurium,12 Streptococcus zooepidemicus, 13 Mycoplasma pneumonias, 14 mycobacterium tuberculosis,15 Varicella zoster virus, 16 human immunodeficiency virus (HIV), 17,18 Ade-novirus,19 and others . 20 Suggested mechanisms by which infection may cause DVT include local invasion by the infectious agent in the surrounding tissues, induc-tion of inflammation by the infectious process in the adjacent tissues, direct endothelial damage induced by the infectious agent, and induction of a transient hypercoagulable state. 12,16,17,20 Indeed, protein S deficiency with DVT was observed during infection with Salmonella typhimurium,12 and HIV.17 In our patient, protein C, protein S, antithrombin III, and APCR activity were normal, and antiphospholipid antibodies were not detected.…”

Section: _ Conclusion 'mentioning

confidence: 99%

Deep Venous Thrombosis Associated with Acute Brucellosis

Odeh

Pick²,

Oliven³

2000

Angiology

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Vascular complications, including arteries and veins associated with Brucella infection, have rarely been reported. To date, only three cases of deep venous thrombosis (DVT) of the lower extremities associated with brucellosis have been previously reported. The authors describe another case of DVT of the right leg in association with acute Brucella infection. Since infection with Brucella may be asymptomatic, and the manifestations of acute brucellosis are exceedingly nonspecific, and in view of the potential for Brucella to cause unexplained or unusual illness involving almost any organ including blood vessels, the authors' case and those previously reported suggest that brucellosis should be included among the causes and infections taken into account in patients suffering from DVT, particularly in those coming from Brucella-endemic areas.

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Acquired deep venous thrombosis in children

Cited by 8 publications

References 27 publications

Prevalence of deep venous thrombosis in the lower extremities of children in the intensive care unit

Prevalence of deep venous thrombosis in the lower extremities of children in the intensive care unit

Occlusive Thrombus Formation on Indwelling Catheters: In Vitro Investigation and Computational Analysis

Deep Venous Thrombosis Associated with Acute Brucellosis

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