“…Between 40 and 50 days after infection, during what is considered the chronic phase, neurological signs are a consequence of demyelination (Martella et al, 2008). In this process, cytotoxic CD8+ T cells activated by proinflammatory cytokines, such as interleukin-6, IL-8, tumor necrosis factor, and interleukin-12 (Beineken et al, 2009;Seehusen et al, 2016), infiltrate the myelin sheath, causing its destruction, resulting inimpaired nerve signal conduction, movements, cognition, and other functions, depending on the region involved, which may provoke paresis, paralysis, myoclonus, fasciculation, involuntary jaw movement, and convulsions in these patients.…”