Abstract:Introduction:
Novel cancer therapies leading to increased survivorship of cancer patients have been negated by a concomitant rise in cancer therapies-related cardiotoxicities. Sunitinib, a multi receptor tyrosine kinase inhibitor, has been reported to cause vascular toxicity. Long non-coding RNAs (lncRNAs) are emerging regulators of biological processes in endothelial cells (ECs); however, their role in cardio-oncology remains poorly understood.
Hypothesis:
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