“…Idiopathic form is usually observed as accompanied to embryological anomalies such as greater omentum hypoplasia and mesenteric vessel malformation. Secondary form, on the other hand, could arise as a result of immunological diseases (SLE and sarcoidosis), infections (tuberculosis), cirrhosis, liver plantation, ventriculoperitoneal shunts, chronic ambulatory peritoneal dialysis, intravenous medicine administration, gastrointestinal malignancies, intra-abdominal surgery (abdominal washing using povidone), beta-adrenergic blocker use (proctolol), familial Mediterranean fever, protein S failure, and gynecological diseases (uterus leiomyoma, endometriotic cysts, luteinized ovarian thecoma, and ruptured dermoid cyst) [4,[7][8][9]. Various acquired factors play a role in its etiology, but the common feature is the development of inflammatory reaction inside the abdomen [1].…”