A topographical study of increased vascular permeability in passive cutaneous anaphylaxis in the chicken

Pillai, A. G. R.; Awadhiya, R.P.; Vegad, J. L.

doi:10.1007/bf00570919

Cited by 9 publications

(4 citation statements)

References 15 publications

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“…This is similar to the response obtained in PCA (Pillai et al, 1987b). A similar, though stronger, biphasic pattern of increased vascular permeability was also observed by Awadhiya et al (1980a) in the chicken mesenteric vessels following intraperitoneal injection of turpentine.…”

Section: Discussionsupporting

confidence: 90%

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Pathology of passive mesenteric anaphylaxis in the chicken

Pillai¹,

Awadhiya²,

Vegad³

1988

Avian Pathology

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Section: Discussionsupporting

confidence: 90%

“…Similar venular response was also observed in PCA (Pillai et al, 1987b). Histamine, 5-hydroxytryptamine and bradykinin have been found to induce an increase in vascular permeability in the venules only (Awadhiya et al, 1980b).…”

Section: Discussionsupporting

confidence: 73%

Pathology of passive mesenteric anaphylaxis in the chicken

Pillai¹,

Awadhiya²,

Vegad³

1988

Avian Pathology

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“…Venule-specific inflammatory responses have been observed in various mammalian organs including cremaster muscle (59), diaphragm (94), trachea (9,62), cheek pouch (103), mesentery (29,46,54,72,118), skin (44,71,78), bladder (91), stomach (27,68), pancreas (27), and intestine (27).…”

Section: Receptors For Inflammatory Mediatorsmentioning

confidence: 99%

Determinants of Endothelial Cell Phenotype in Venules

Thurston¹,

Bałuk²,

McDonald³

2000

Microcirculation

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Inflammatory stimuli cause plasma leakage and leukocyte adhesion in venules but not in capillaries or arterioles. The specific response of venules is governed by phenotypic specialization of the venular endothelial cells. What regulates this specialized phenotype? Several recent developments have shed new light on this question and may challenge our thinking about regulation of the venular endothelial cell phenotype. In this review, we consider some of the molecular markers of venular endothelial cells, the hemodynamic and molecular factors that may regulate the phenotype of venular endothelial cells, and abnormalities in endothelial cell phenotype in disease-related angiogenesis and microvascular remodeling. The expanding list of molecular markers may help clarify the physiologic and molecular factors that regulate the phenotype of venular endothelial cells in normal development and disease.

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“…A number of different stimuli have been employed, namely, chemical (Carlson & Allen, 1969;Jortner & Adams, 1971;Nair, 1973;Stadecker et ah, 1977;Awadhiya et al 1980aAwadhiya et al ,b, 1981aJain et al, 1982;Ito & Bohm, 1986;Sinha et al, 1987aSinha et al, ,b, 1988Ito et al, 1989), thermal (Awadhiya et al, 1981b), bacterial (Carlson & Allen, 1969;Nair, 1973), parasitic (Nair, 1973) and immunological (Chand et al, 1976;Rose and Bradley, 1977;Awadhiya et al, 1982;Dhodapkar et al, 1982;1983,1984Pillai et al, 1987;1988a,b,c,d). However, no information is available on the pathology of inflammatory-reparative response in wound healing in the chicken.…”

Section: Introductionmentioning

confidence: 99%

Pathology of inflammatory‐reparative response in punched wounds of the chicken skin

Katiyar¹,

Vegad²,

Awadhiya³

1992

Avian Pathology

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SUMMARYVascular and cellular responses were examined sequentially in punched wounds of the chicken skin. The 'dye' and the 'colloidal carbon' technique revealed that the increased permeability response was immediate and sustained. The response attained its peak at 1 hour and persisted for up to 6 hours, but the increased permeability was prolonged over a period of 48 h. The increase in vascular permeability at all stages was confined to venules and small veins only. In the later stages of healing, the newly formed vessels in the granulation tissue were leaky.The initial migration of leukocytes comprised heterophils and monocytoid cells. This was soon followed by infiltration of some basophils. A striking feature of the reaction, in the granulation tissue, was that the monocytoid cells apparently coalesced, formed syncytia, and even what appeared to be distinct multinucleated giant cells. Whereas mast cells participated in the early stages, they were absent in the granulation tissue. Proliferation of the fibroblasts began around 18 h, angiogenesis and re-epithelialization at 3 days, and the healing was complete by about 7 days.

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A topographical study of increased vascular permeability in passive cutaneous anaphylaxis in the chicken

Cited by 9 publications

References 15 publications

Pathology of passive mesenteric anaphylaxis in the chicken

Pathology of passive mesenteric anaphylaxis in the chicken

Determinants of Endothelial Cell Phenotype in Venules

Pathology of inflammatory‐reparative response in punched wounds of the chicken skin

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