A review of chemical control measures for porina

Waller, JM

doi:10.30843/nzpp.1968.21.10469

Cited by 2 publications

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“…comms) suggest a sex ratio of 1:1 for W. cervinata. Although larvae cannot be sexed, the difference in size between the sexes as adults and pupae (Waller 1966) must pre-exist in larvae, at least during the later instars, such that weight allocation caused the heaviest groups to be predominantly female. That there were no differences in susceptibility between groups suggests that larval gender does not affect susceptibility.…”

Section: Weight Maturity and Gendermentioning

confidence: 99%

Susceptibility to three organophosphorus insecticides of Wiseana spp. larvae (Lepidoptera: Hepialidae)

Perrott¹

1974

New Zealand Journal of Zoology

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When test groups of Wiseana larvae were standardised by weight, estimates of susceptibility to fenitrothion applied topically in 1-µlitre volumes of isopropyl alcohol to batches of 10 showed no essential differences between the populations examined, nor between larvae in the weight range 85-1177 mg. Mean LD 50 values for fenitrothion (11 larval populations) and for diazinon and trichlorfon (3 larval populations) were 0.84, 2.31, and 1.39 µg/g respectively. Single LD 50 estimates for the same toxicants ingested on clover leaflets were 1.29, 4.57, and 1.5 µg/g respectively. The close relationship between weight and susceptibility would allow small batches of larvae of almost any weight to be used for the detection of resistance, for which the following discriminating doses (based on 2 × LD 100 in µg/g) are proposed: fenitrothion 3.0; diazinon 8.0; trichlorfon 5.0.

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Section: Weight Maturity and Gendermentioning

confidence: 99%

Susceptibility to three organophosphorus insecticides of Wiseana spp. larvae (Lepidoptera: Hepialidae)

Perrott¹

1974

New Zealand Journal of Zoology

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Inherited Metabolic Diseases of the Nervous System

Brady

1976

Science

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This overview was designed primarily to provide examples of hereditary metabolic disorders that result in nervous system dysfunction. Some of the more frequently encountered pathological conditions were selected in order to illustrate the mechanisms and the consequences of the metabolic derangements. Therapeutic approaches for the correction of such disorders are discussed where it appears appropriate. In time the precise etiology for those eponymous genetic conditions with stereotyped pathologic and clinical manifestations such as Huntington's chorea (79) and Friedreich's ataxia (80) will be disclosed. It is possible that some forms of epilepsy (81) and perhaps certain psychiatric disturbances (82) will be shown to be inherited metabolic disorders. As our knowledge and skill increase, this logic may eventually be extended to biochemical explanations of variation in individual skills and talents. Certainly innovative extrapolation and novel research directions will be necessary to provide an understanding of these differences. However, it is axiomatic in research that each useful contribution serves largely as a point of departure for further accomplishments.

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A review of chemical control measures for porina

Cited by 2 publications

References 0 publications

Susceptibility to three organophosphorus insecticides of Wiseana spp. larvae (Lepidoptera: Hepialidae)

Susceptibility to three organophosphorus insecticides of Wiseana spp. larvae (Lepidoptera: Hepialidae)

Inherited Metabolic Diseases of the Nervous System

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