A Report of Toxin‐Induced Graft Injury Following Liver Transplantation for Yellow Phosphorus Poisoning

Reply

“…We read the article by McMahon et al with great interest. [1] The authors found that HBV genotype was strongly associated with HCC incidence in the Hepatitis B Alaska cohort and suggested that HBV genotype should be considered in risk factor stratification of HCC. [1] Although inspiring, several issues warrant further clarification.…”

“…Ours was the first such report of YP‐induced allograft injury, a hitherto unknown entity. [ 1 ] Reports such as ours help highlight the rare causes of EAD, helping future clinicians think of this as a possible cause. We concur that this topic has large gray areas, leaving a tremendous scope for further research, including those regarding the nuances of post‐LT management.…”

“…As stated in our article, these findings were very similar to the explant liver histology (Figure 1B,D). [ 1 ]…”

Letter to the Editor: Toxin‐induced graft injury following liver transplantation for yellow phosphorus poisoning

“…As stated in our article, these findings were very similar to the explant liver histology (Figure 1B,D). [1] The authors' premise that 70% of the body stores of YP accumulates in the liver is factually inaccurate. Eighty-five percent of the stores are in fact in the bones, muscles, and fat.…”

“…To the editor, We read with interest the article by Jasper et al [1] regarding toxin-induced liver injury (TLI) after liver transplantation (LT) in yellow phosphorus (YP) poisoning. The authors postulate that early allograft dysfunction (EAD) is secondary to TLI from YP released from nonhepatic tissues after LT. We believe that there are theoretical and practical problems with this hypothesis.…”