2004
DOI: 10.1016/j.cub.2004.11.039
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A marker for the end of adolescence

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Cited by 1,263 publications
(1,195 citation statements)
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References 17 publications
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“…The movement away from Morningness associated with increasing age was reliable, F(3, 255) = 5.30, p = . 001, ν 2 = .06, replicating other findings (e.g., Kim et al, 2002;Roenneberg et al, 2004).…”
Section: Chronotype Preferencessupporting
confidence: 85%
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“…The movement away from Morningness associated with increasing age was reliable, F(3, 255) = 5.30, p = . 001, ν 2 = .06, replicating other findings (e.g., Kim et al, 2002;Roenneberg et al, 2004).…”
Section: Chronotype Preferencessupporting
confidence: 85%
“…The mechanisms that underlie the adolescent shift away from Morningness may have environmental, social, and biological underpinnings (Carskadon et al, 1993;Kim et al, 2002;Roenneberg et al, 2004). Whatever the source of this shift, our evidence suggests that Evening-type adolescents are far more likely than Morning-types to fall into the borderline/ clinical category of the most widely used behavioral assessment instrument.…”
Section: Discussionmentioning
confidence: 79%
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“…The MCTQ is used to calculate the MSF as the mid-point between sleep onset and sleep end. MSF was corrected for oversleep on free days (MSF sc : Mid Sleep point on Free days, Sleep Corrected) that occurs as a result of sleep debt [MSF sc  = MSF – (SDf – ((((nWD × SDw) + (7 − nWD)) × SDf)/7)), where SDf is the sleep duration of free days, SDw is the sleep duration of workdays and nWD is the number of workdays (Roenneberg et al 2004). In cases where the numbers of workdays were missing, five workdays were assigned.…”
Section: Methodsmentioning
confidence: 99%
“…a delay in the secretion of endogenous markers of circadian rhythmicity), with a peak (and thus the latest sleep time) at around the age of 22 years. This peak is thought to be a biological marker of the end of adolescence (Roenneberg et al 2004). Rather than simply being a lengthening of the circadian period in adolescents, this phase delay is considered to be the result of a reorganisation of the circadian system and its interaction with homeostatic sleep pressure (Carskadon 2008;Hagenauer et al 2009).…”
mentioning
confidence: 99%