A Glimpse of Some Unpublished Santayana Manuscripts

Grossman, Morris

doi:10.2307/2022990

Cited by 2 publications

(3 citation statements)

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“…However, the exponential dependence on m* L and V* should still be reflected in a more detailed treatment. This was recently demonstrated in a numerical study extending the calculations of [44]; in the localized regime, the probability to find transmission trajectory with value smaller than a given γ is exponential with W 1/2 and L [45].…”

Section: Dynamics In the Dead-woodmentioning

confidence: 64%

Slow dynamics of electron glasses: The role of disorder

Ovadyahu

2017

Phys. Rev. B

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We examine in this work the role of disorder in contributing to the sluggish relaxation observed in intrinsic electron-glasses. Our approach is guided by several empirical observations: First and foremost, Anderson localization is a pre-requisite for observing these nonequilibrium phenomena. Secondly, sluggish relaxation appears to favor Anderson-insulators with relatively large Fermi-energies (hence proportionally large disorder). These observations motivated us to consider a way to measure the underlying disorder in a realistic Anderson insulator. Optical study using a series of amorphous indium-oxide (InxO) establish a simple connection between carrier-concentration and the disorder necessary to approach the metal-insulator transition from the insulating side. This is used to estimate the typical magnitude of the quenched potential-fluctuation in the electron-glass phase of this system. The implications of our findings on the slow dynamics of Anderson-insulators are discussed. In particular, the reason for the absence of a memory-dip and the accompanying electron-glass effects in lightly-doped semiconductors emerges as a natural consequence of their weak disorder.

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Section: Dynamics In the Dead-woodmentioning

confidence: 64%

Slow dynamics of electron glasses: The role of disorder

Ovadyahu

2017

Phys. Rev. B

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“…Unfortunately, there occasionally are patients with precisely the reverse findings. Severe mitral regurgitation may lead to the appearance of fully oxygenated blood in the distal pulmonary artery segments, presumably reflecting regurgitation of blood retrograde through the pulmonary capillaries and into the pulmonary artery [8]. While careful catheter placement under fluoroscopic guidance should prevent confusion by ensuring that blood samples for oximetry are drawn from the main pulmonary artery (rather than the segmental branches of the pulmonary arteries), many of the studies are done without such fluoroscopic control.…”

Section: Discussionmentioning

confidence: 99%

“…However, several technical considerations made it difficult in occasional patients to differentiate this defect from acute mitral regurgitation due to papillary muscle dysfunction or rupture, the other major cause of a new murmur appearing after infarction [8,9]. Furthermore, the status of overall and regional left ventricular function cannot be ascertained from a right heart catheterization alone, requiring left ventricular angiography [7].…”

Section: Introductionmentioning

confidence: 99%

Radionuclide Scintigraphy in the Evaluation of Ventricular Septal Defect Complicating Acute Myocardial Infarction

Wynne

Fishbein

Holman

et al. 1978

Catheterization and Cardiovascular Diagnosis

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A 67-year-old man with an autopsy-proven ventricular septal defect (VSD) following an acute inferoposterior myocardial infarction underwent bedside evaluation by radionuclide imaging techniques. The presence of the VSD was confirmed, and the size of the shunt determined by qualitative and quantitative radionuclide angiocardiography. In addition, radionuclide ventriculography permitted assessment of global as well as regional left ventricular function.

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A Glimpse of Some Unpublished Santayana Manuscripts

Cited by 2 publications

References 0 publications

Slow dynamics of electron glasses: The role of disorder

Slow dynamics of electron glasses: The role of disorder

Radionuclide Scintigraphy in the Evaluation of Ventricular Septal Defect Complicating Acute Myocardial Infarction

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