A Family History Study of Borderline Subtypes

Schulz, Patricia M.; Soloff, Paul H.; Kelly, Thomas P.; Morgenstern, Merle; Franco, Robert Di; Schulz, S. Charles

doi:10.1521/pedi.1989.3.3.217

Cited by 56 publications

(28 citation statements)

References 2 publications

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“…One study that found a heightened prevalence of schizophrenia in relatives did not use the DSM-III borderline criteria in the probands or family members but relied on Kernberg's criteria, making it difficult to compare this study with the others [49]. Additionally, most of the studies reporting an increased prevalence of psychotic disorders in relatives of BPD individuals did not control for schizotypal participants in the borderline sample [49,50]. This suggests that it is the comorbidity of BPD and schizotypal personality Fig.…”

Section: Association Between Maltreatment In Childhood and Psychotic mentioning

confidence: 99%

Borderline Personality Disorder and Psychosis: A Review

Barnow

Arens

Sieswerda

et al. 2010

Curr Psychiatry Rep

103

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Early views of borderline personality disorder (BPD) were based on the idea that patients with this pathology were "on the border" of psychosis. However, more recent studies have not supported this view, although they have found evidence of a malevolent interpersonal evaluation and a significant proportion of BPD patients showing psychotic symptoms. For example, in one study, 24% of BPD patients reported severe psychotic symptoms and about 75% had dissociative experiences and paranoid ideation. Thus, we start with an overview regarding the prevalence of psychotic symptoms in BPD patients. Furthermore, we report findings of studies investigating the role of comorbidity (eg, post-traumatic stress disorder) in the severity and frequency of psychotic symptoms in BPD patients. We then present results of genetic and neurobiological studies comparing BPD patients with patients with schizophrenia or nonschizophrenic psychotic disorders. In conclusion, this review reveals that psychotic symptoms in BPD patients may not predict the development of a psychotic disorder but are often permanent and severe and need careful consideration by clinicians. Therefore, adequate diagnosis and treatment of psychotic symptoms in BPD patients is emphasized.

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Section: Association Between Maltreatment In Childhood and Psychotic mentioning

confidence: 99%

Borderline Personality Disorder and Psychosis: A Review

Barnow

Arens

Sieswerda

et al. 2010

Curr Psychiatry Rep

103

View full text Add to dashboard Cite

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“…The difference of social class between borderline and Axis I1 PD controls attained statistical significance across 215 studies in that cluster. Average differences of social class attained statistical significance in 3/5 pairwise contrasts which represented data derived from four studies within the Axis I1 PD cluster (Edell, 1987;Schulz et al, 1989;Zanarini et al, 1987Zanarini et al, , 1988. Two outcomes arising from these contrasts were in the predicted direction.…”

Section: Social Classmentioning

confidence: 87%

“…The findings were compared statistically with either social class or educational background or both variables in (a) community residents of Durham, North Carolina (Swartz et al, 1990); (b) European countries such as Italy and Norway (Alnaes & Torgersen, 1988;Bellodi et al, 1992;Dahl, 1986); (c) diagnoses of borderline personality according to the DIB; (d) Borderline Personality Scale; and (e) DSM-111-R criteria. Patients in 4/11 studies were evaluated for the diagnoses of borderline PD according to either the DIB or revised version (DIB-R) (Clarkin, Widiger, Frances, Hurt & Gilmore, 1983;Schulz et al, 1989;Zanarini et al, 1988Zanarini et al, , 1989. In the study, however, conducted at Western Psychiatric Institute (Schulz et al, 1989), the DIB criteria could not be assessed for convergent validation with the DSM-I11 diagnosis.…”

Section: Sociodemographic Variablesmentioning

confidence: 99%

“…Patients in 4/11 studies were evaluated for the diagnoses of borderline PD according to either the DIB or revised version (DIB-R) (Clarkin, Widiger, Frances, Hurt & Gilmore, 1983;Schulz et al, 1989;Zanarini et al, 1988Zanarini et al, , 1989. In the study, however, conducted at Western Psychiatric Institute (Schulz et al, 1989), the DIB criteria could not be assessed for convergent validation with the DSM-I11 diagnosis.…”

Section: Sociodemographic Variablesmentioning

confidence: 99%

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Sociodemography of Borderline Personality Disorder (Pd): A Comparison with Axis II Pds and Psychiatric Symptom Disorders Convergent Validation

Taub¹

1996

International Journal of Neuroscience

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A theoretical objective of the present meta-analysis based upon data derived from a previously reported review (Taub, 1995), was to test two inductive hypotheses empirically regarding educational background and social class across different criteria for the DSM-III diagnosis of borderline personality disorder (PD). A secondary purpose was to determine whether comorbidity of borderline PD with other Axis II PDs would significantly delineate socioeducational variables. Across 7/8 pairwise contrasts which represented five studies, distribution of Hollingshead Redlich (H-R) social classes II-IV borderline PD (N = 326) significantly exceeded that in 457 diagnostic controls with Axis II PDs and psychiatric symptom disorders. Although average differences, as well as, interactions reflected by values of the H-R two-factor scale attained statistical significance these were were less consistent in magnitude and direction versus outcomes yielded by distribution of social classes. For the borderline PD diagnosis, the inductive hypotheses were substantiated by findings of significantly advanced scholastic achievement, as well as the younger age of most cohorts versus diagnostic controls with Axis II PDs and psychiatric symptom disorders; and in pairwise contrasts of outpatients with hospitalized cohorts. Comorbidity of the borderline PD diagnosis was associated with significantly lower social class, scholastic achievement and to a lesser extent, more severe psychopathology. Evidence for predominantly convergent validation relative to the socioeducational variables was substantiated by comparisons with (a) cohorts selected by criteria of the DSM-III-R, Gunderson's DIB and Borderline Personality Scale: (b) Norwegian females admitted to Gaustad Hospital and (c) patients with the DSM-III diagnosis of borderline PD attending an outpatient clinic in Norway.

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“…BPD and MD patients shared some characteristics of sleep continuity, but the structure of sleep was different in the two groups. We are now comparing BPD to recurrent brief depression and MD with endocrine neuropsychiatric tests and sleep EEG in order to improve our understanding of the biological nature of depressive symptoms in BPD because the link between BPD and the affective disorders is a controversial topic [34,39,46,48,[59][60][61][62][63][64][65].…”

Section: Introductionmentioning

confidence: 99%

Effects of Carbamazepine on Dexamethasone Suppression and Sleep Electroencephalography in Borderline Personality Disorder

Fuente

Bobes²,

Vizuete³

et al. 2002

Neuropsychobiology

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The pathophysiology of borderline personality disorder (BPD) remains obscure, but there is mounting evidence of brain dysfunction without focal abnormality. The dexamethasone suppression test (DST) and sleep electroencephalography (sleep EEG) have been studied in BPD, but the findings seem to be related to a concomitant axis I diagnosis of major depression (MD) rather than to BPD itself. There is no effective treatment for BPD. Carbamazepine (CBZ) has shown contradictory results and in a previous study, our results were negative. In this study, we investigated the effects of CBZ versus placebo on the DST and sleep EEG in a sample of 20 BPD patients without concomitant MD. CBZ given at doses that are therapeutic for epilepsy and affective disorders may have an effect on the DST and sleep EEG in BPD. CBZ significantly increased the postdexamethasone plasma cortisol values. This did not parallel MD or an increase in the Hamilton depression rating scores. CBZ also increased slow wave sleep (SWS). The mechanisms by which CBZ increased postdexamethasone plasma cortisol levels and SWS in BPD are discussed.

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A Family History Study of Borderline Subtypes

Cited by 56 publications

References 2 publications

Borderline Personality Disorder and Psychosis: A Review

Borderline Personality Disorder and Psychosis: A Review

Sociodemography of Borderline Personality Disorder (Pd): A Comparison with Axis II Pds and Psychiatric Symptom Disorders Convergent Validation

Effects of Carbamazepine on Dexamethasone Suppression and Sleep Electroencephalography in Borderline Personality Disorder

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