A computer program for ECG classification according to the minnesota code

Duisterhout, J. S.; May, Jon; Herpen, Gerard van; Distelbrink, C A; Talmon, Jan L.; Plokker, H.W.M.

doi:10.1016/s0022-0736(77)80012-5

Cited by 5 publications

(1 citation statement)

References 14 publications

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“…A number of previous studies suggested that increased heterogeneity of ventricular repolarization is an important determinant of ventricular tachyarrhythmias 1–4 . The variability of QT interval in 12‐lead ECG, i.e., QT dispersion, which is thought to reflect a regional difference in the repolarization process, 1 , 5 has aroused considerable interest in relation to its arrhythmogenicity, 6–10 and has been used as a noninvasive predictor of clinical outcome in patients with congenital long‐QT syndrome, 1 , 11,12 vasospastic angina, 13 myocardial infarction, 2 , 14–18 chronic congestive heart failure 19,20 and dilated cardiomyopathy, 21,22 etc.…”

Section: Introductionmentioning

confidence: 99%

Determinant of QT Dispersion in Patients with Hypertrophic Cardiomyopathy

Kawasaki¹,

Azuma²,

Kuribayashi³

et al. 2003

Pacing Clinical Electrophis

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QT dispersion is thought to reflect a regional difference in repolarization process although QT interval is composed of depolarization and repolarization. This study was designed to investigate the effect of depolarization and repolarization on QT dispersion in hypertrophic cardiomyopathy. Standard 12-lead ECG was recorded in 70 hypertrophic cardiomyopathy patients with anteroseptal wall hypertrophy (HC-As), 8 patients with lateral wall hypertrophy (HC-L), 8 patients with diffuse hypertrophy (HC-D), and 46 normal controls. QRS, JTc, maximum and minimum QTc, and QTc dispersion were compared. The maximum QTc was greater in HC-As and HC-L than in the control; the minimum QTc was similar in all 3 groups; consequently, QTc dispersion was greater in HC-As and HC-L. In HC-D, the maximum QTc and the minimum QTc were greater than the control, which produced QTc dispersion similar to that in the control. JTc did not differ among 4 groups. In hypertrophic cardiomyopathy, both QTc and QRS duration were increased in the leads coinciding with the left ventricular portion of localized hypertrophy. We conclude that QTc dispersion depended on the heterogeneity of QRS duration or depolarization rather than repolarization, which in fact may be ascribed to the regionally different hypertrophy of the left ventricle in hypertrophic cardiomyopathy.

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