2008
DOI: 10.1016/j.jns.2008.03.005
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A case of Vernet syndrome with varicella zoster virus infection

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Cited by 19 publications
(12 citation statements)
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References 3 publications
(6 reference statements)
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“…In Wang’s case, 4 hypoperistalsis occurred in the middle part of the patient’s esophagus, which convinced the authors that dysphagia was caused by the violation of sympathetic nerves and the parasympathetic nerves’ visceral branch. In the other two cases, Ramsay–Hunt syndrome was caused by the violation of glossopharyngeal nerve 5,6 . The laryngoscope revealed a left vocal cord paralysis, in accordance with the patient’s prominent hoarseness and speechless.…”
mentioning
confidence: 71%
“…In Wang’s case, 4 hypoperistalsis occurred in the middle part of the patient’s esophagus, which convinced the authors that dysphagia was caused by the violation of sympathetic nerves and the parasympathetic nerves’ visceral branch. In the other two cases, Ramsay–Hunt syndrome was caused by the violation of glossopharyngeal nerve 5,6 . The laryngoscope revealed a left vocal cord paralysis, in accordance with the patient’s prominent hoarseness and speechless.…”
mentioning
confidence: 71%
“…There are few case reports of varicella-zoster virus as a rare cause of Vernet syndrome [8,9]. The jugular foramen is divided into two parts?…”
Section: Discussionmentioning
confidence: 99%
“…Thus, it is unclear whether the left jugular foramen size contributed to Vernet syndrome in the present case, and further research is needed. The general prognosis of Vernet syndrome is unknown; however, treating Vernet syndrome with combined therapy with antiviral agents and steroids has shown a good prognosis showing restored muscle strength in shoulder joint abduction with improved dysphagia and hoarseness [8,9]. …”
Section: Discussionmentioning
confidence: 99%
“…[2][3][4] This condition has been termed zoster sine herpete. A wide spectrum of neurologic disorders that include polyneuritis cranialis, aseptic meningitis, encephalitis, acute polyneuritis, and myelitis are associated with VZV infection without skin lesions.…”
Section: Discussionmentioning
confidence: 99%
“…[3][4][5] The cause for these clinicopathologic diversities is unclear. 1,2 However, some D178N patients who had homozygosity for methionine at codon 129 (D178N-129MM) were reported re-cently to have the CJD phenotype.…”
Section: Discordant Clinicopathologic Phenotypes In a Japanese Kindrementioning
confidence: 99%