A case of rheumatoid arthritis presenting with postherpetic neuralgia and abdominal-wall pseudohernia

Dobrev, Hristo; Atanassova, Penka A.; Sirakov, Vladimir N.; Zisova, Lilia G

doi:10.1007/s00296-009-1360-2

Cited by 11 publications

(11 citation statements)

References 7 publications

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“…In 1885, Taylor [15] reported the first known case of a patient with a segmental zoster abdominal paresis. Since that time, other similar cases have been described [10‐13,15‐45]. The aim of this article was to review the existing literature with regard to this specific complication and to summarize the findings from the published evidence.…”

Section: Introductionmentioning

confidence: 93%

Segmental Zoster Abdominal Paresis (Zoster Pseudohernia): A Review of the Literature

Chernev

Dado

2013

PM&R

130

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Herpes zoster is a distinct clinical syndrome that may present with a segmental zoster paresis. Although thoracic dermatomes are the most commonly affected, paresis of the abdominal muscles has been less frequently reported. To review the existing published evidence regarding this unusual clinical entity, a literature search of PubMed and Google was performed. In total, 35 articles that described 36 individual cases were identified. The information from all the cases was tabulated for the analysis. The mean age was 67.5 years. The ratio of men to women was approximately 4:1. The left and right side were approximately equally affected. The most affected associated dermatome was T11. In 88.9% of the patients, the typical herpetic rash preceded the abdominal weakness. The mean latent period from rash to onset of abdominal muscle weakness was 3.5 weeks. Electrodiagnostic studies confirmed the diagnosis in 95% of the tested patients. Complete recovery with conservative measures occurred in 79.3% of the patients who were followed-up for recovery, with a mean time for recovery of 4.9 months. Visceral neuropathy co-occurred in 19.4% of the patients. Because of its self-limited nature and good prognosis, recognition of this complication is important to prevent unnecessary diagnostic studies and procedures. Electrodiagnostic studies can be effectively used to confirm the diagnosis. Because visceral neuropathy commonly co-occurs with segmental zoster abdominal paresis, it should be actively investigated and treated.

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Section: Introductionmentioning

confidence: 93%

Segmental Zoster Abdominal Paresis (Zoster Pseudohernia): A Review of the Literature

Chernev

Dado

2013

PM&R

130

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“…HZ results from the reactivation of the neurotropic varicella‐zoster virus (VZV), latent after primary infection, in the dorsal root ganglia of the spinal cord . This virus, like the other Herpesviridae family viruses, has lifetime latency, although it has a variable period (usually lasting several years after the primary VZV infection) before it reactivates like HZ .…”

Section: Discussionmentioning

confidence: 99%

“…Postherpetic pseudohernia is a rare complication present in 2% of HZ patients . It may coincide with vesicular eruption or occur shortly after its resolution .…”

Section: Discussionmentioning

confidence: 99%

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Postherpetic abdominal pseudohernia. Presentation of a clinical case and literature review

2018

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“…It is sometimes referred to as a "pseudohernia." 6,7,16,21 One of the main causes of abdominal wall paresis is iatrogenic injury during surgery. It has been reported after abdominal and pelvic surgery.…”

Section: Discussionmentioning

confidence: 99%

Abdominal wall paresis as a complication of minimally invasive lateral transpsoas interbody fusion

Dakwar

Baaj

et al. 2011

FOC

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Object The minimally invasive lateral transpsoas approach for interbody fusion has been increasingly employed to treat various spinal pathological entities. Gaining access to the retroperitoneal space and traversing the abdominal wall poses a risk of injury to the major nervous structures. Nerve injury of the abdominal wall can potentially lead to paresis of the abdominal musculature and bulging of the abdominal wall. Abdominal wall nerve injury resulting from the minimally invasive lateral retroperitoneal transpsoas approach has not been previously reported. The authors describe a case series of patients presenting with paresis and bulging of the abdominal wall after undergoing a minimally invasive lateral retroperitoneal approach. Methods The authors retrospectively reviewed all patients who underwent a minimally invasive lateral transpsoas approach for interbody fusion and in whom development of abdominal paresis developed; the patients were treated at 4 institutions between 2006 and 2010. All data were recorded including demographics, diagnosis, operative procedure, positioning, hospital course, follow-up, and complications. The onset, as well as resolution of the abdominal paresis, was reviewed. Results The authors identified 10 consecutive patients in whom abdominal paresis developed after minimally invasive lateral transpsoas spine surgery out of a total of 568 patients. Twenty-nine interbody levels were fused (range 1–4 levels/patient). There were 4 men and 6 women whose mean age was 54.1 years (range 37–66 years). All patients presented with abdominal paresis 2–6 weeks postoperatively. In 8 of the 10 patients, abdominal wall paresis had resolved by the 6-month follow-up visit. Two patients only had 1 and 4 months of follow-up. No long-term sequelae were identified. Conclusions Abdominal wall paresis is a rare but known potential complication of abdominal surgery. The authors report the first case series associated with the minimally invasive lateral transpsoas approach.

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A case of rheumatoid arthritis presenting with postherpetic neuralgia and abdominal-wall pseudohernia

Cited by 11 publications

References 7 publications

Segmental Zoster Abdominal Paresis (Zoster Pseudohernia): A Review of the Literature

Segmental Zoster Abdominal Paresis (Zoster Pseudohernia): A Review of the Literature

Postherpetic abdominal pseudohernia. Presentation of a clinical case and literature review

Abdominal wall paresis as a complication of minimally invasive lateral transpsoas interbody fusion

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