“…Fistula formation into other organs can occasionally occur in IPMN [4][5][6][7][8][9][10][11]. In the present study, among the IPMN patients who had been diagnosed by ERCP and EUS, fistula formation was observed in 6.6%.…”
Section: Discussionmentioning
confidence: 51%
“…Characteristic clinicopathological features of IPMN are (1) secretion of a large quantity of mucin by papillary neoplasms, (2) dilatation of the main pancreatic duct (MPD) or branches, (3) slow growth with a favorable prognosis, (4) intraepithelial spread along the pancreatic duct, (5) rarity of symptoms, (6) possible multicentric development of cancer in the whole pancreas, (7) occasional concomitance of common-type invasive cancer of the pancreas, and (8) fistula formation to other organs [3].…”
There were 2 processes in the development of fistulas in IPMN. Of those patients showing fistula formation, 94% had intestinal-type IPMN, and 67% showed mechanical penetration. Delineation of a mass with the mixed-echo pattern suggested an invasive penetration due to colloid carcinoma.
“…Fistula formation into other organs can occasionally occur in IPMN [4][5][6][7][8][9][10][11]. In the present study, among the IPMN patients who had been diagnosed by ERCP and EUS, fistula formation was observed in 6.6%.…”
Section: Discussionmentioning
confidence: 51%
“…Characteristic clinicopathological features of IPMN are (1) secretion of a large quantity of mucin by papillary neoplasms, (2) dilatation of the main pancreatic duct (MPD) or branches, (3) slow growth with a favorable prognosis, (4) intraepithelial spread along the pancreatic duct, (5) rarity of symptoms, (6) possible multicentric development of cancer in the whole pancreas, (7) occasional concomitance of common-type invasive cancer of the pancreas, and (8) fistula formation to other organs [3].…”
There were 2 processes in the development of fistulas in IPMN. Of those patients showing fistula formation, 94% had intestinal-type IPMN, and 67% showed mechanical penetration. Delineation of a mass with the mixed-echo pattern suggested an invasive penetration due to colloid carcinoma.
This case highlights that a pancreatogastric fistula can develop after acute inflammation of the pancreas in the absence of cancer invasion. Further information regarding IPMN-associated fistulae is necessary to clarify the pathogenesis, diagnosis, appropriate surgical intervention and prognosis for this disorder.
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