A Case of Hyporeninemic Hypoaldosteronism in the Dog

Kreissler, J.J.; Langston, Cathy

doi:10.1111/j.1939-1676.2011.0741.x

Cited by 16 publications

(11 citation statements)

References 18 publications

(22 reference statements)

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“…Furthermore, hypoadrenocorticism was later ruled out by ACTH stimulation testing. Primary hypoaldosteronism without hypocortisolism has been reported in dogs but in this patient hypoaldosteronism and hyperkalemia resolved after discontinuation of heparin treatment.…”

Section: Case Summarymentioning

confidence: 72%

Heparin‐Induced Hyperkalemia in a Dog Receiving Continuous Renal Replacement Treatment

Geesaman

Bach

Monaghan³

2015

Veterinary Internal Medicne

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Section: Case Summarymentioning

confidence: 72%

Heparin‐Induced Hyperkalemia in a Dog Receiving Continuous Renal Replacement Treatment

Geesaman

Bach

Monaghan³

2015

Veterinary Internal Medicne

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“…It has been hypothesized that aldosterone concentrations in these dogs are normal due to sparing of the ZG; however, in most reported cases, the aldosterone concentration was not directly measured. Isolated case reports of hyporeninemic hypoaldosteronism in a dog with renal failure and hyperreninemic hypoaldosteronism in a dog that also had a heart base chemodectoma have been reported in the veterinary literature, but both syndromes are exceedingly rare (Lobetti, 1998;Kreissler and Langston, 2011). 12-5;Baumstark et al, 2014).…”

Section: Glucocorticoid Deficiency Versus Mineralocorticoid Deficiencymentioning

confidence: 99%

Hypoadrenocorticism

Scott‐Moncrieff¹

2015

Canine and Feline Endocrinology

116

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“…This is supported by the finding of normal [Na + ] in euvolemic human patients with primary hypoaldosteronism. A case of hyporeninemic hypoaldosteronism has also been reported in a dog . In this dog, serum potassium concentration was increased, and serum [Na + ] was normal, despite an increased urinary fractional excretion of Na + .…”

Section: Diagnosismentioning

confidence: 77%

Hyponatremia in dogs and cats

Burton

Hopper

2019

J Vet Emergen Crit Care

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Objective: To review the pathophysiology, diagnostic approach, and treatment recommendations for hyponatremia in dogs and cats. Etiology: Hyponatremia almost always results from an increase in total body water (TBW), and not from loss of sodium. Abnormalities in antidiuretic hormone (ADH) are commonly part of the etiology of hyponatremia. Diagnosis: Diagnosis of hyponatremia focuses on the cause of the increase of TBW. Assessment of the patient's volume status and measurement of urine sodium concentration are important factors. Measurement or calculated estimation of plasma osmolality can also guide the assessment of hyponatremia.Therapy: Too rapid correction of serum sodium can precipitate osmotic demyelination syndrome.As a general rule, serum sodium concentration should be raised ≤10 mmol/L over 24 hours, but rapid increases in serum sodium are indicated if neurologic abnormalities are evident. Serum sodium can be increased using hypertonic saline, with dosing based on the patient's calculated sodium deficit. Treatment of the underlying cause of water ingestion or retention is also required to fully resolve hyponatremia. Prognosis:Mortality rates are significantly higher in dogs and cats with hyponatremia compared to those with normal serum sodium concentrations, even in patients with mild hypontremia (<5 mmol/L below the lower value of the reference interval). Hyponatremia is also associated with increased risk of death if present during specific disease states in dogs. K E Y W O R D Santidiuretic hormone, electrolyte, osmolality, sodium

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A Case of Hyporeninemic Hypoaldosteronism in the Dog

Cited by 16 publications

References 18 publications

Heparin‐Induced Hyperkalemia in a Dog Receiving Continuous Renal Replacement Treatment

Heparin‐Induced Hyperkalemia in a Dog Receiving Continuous Renal Replacement Treatment

Hypoadrenocorticism

Hyponatremia in dogs and cats

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