A Case of Celiac Disease

Gweon, Tae‐Geun; Lim, Chul-Hyun; Byeon, Seoug-Wook; Baeg, Myong Ki; Lee, Jong-Yul; Moon, Seok Whan; Kim, Jin-Su; Choi, Myung‐Gyu

doi:10.4166/kjg.2013.61.6.338

Cited by 16 publications

(11 citation statements)

References 8 publications

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“… 69 A case of celiac disease was also reported from Korea. 70 Similar biopsy-positive Asian Canadians with celiac disease were previously noted, including a Chinese woman. 71 A recent extensive study of HLA-haplotypes and wheat consumption in different regions of China also suggested that celiac disease occurs more frequently in China than currently reported.…”

Section: Emerging Risk Estimatessupporting

confidence: 61%

Celiac Disease: A Disorder Emerging from Antiquity, Its Evolving Classification and Risk, and Potential New Treatment Paradigms

Freeman¹

2015

Gut Liver

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Celiac disease is a chronic genetically based gluten-sensitive immune-mediated enteropathic process primarily affecting the small intestinal mucosa. The disorder classically presents with diarrhea and weight loss; however, more recently, it has been characterized by subclinical occult or latent disease associated with few or no intestinal symptoms. Diagnosis depends on the detection of typical histopathological biopsy changes followed by a gluten-free diet response. A broad range of clinical disorders may mimic celiac disease, along with a wide range of drugs and other therapeutic agents. Recent and intriguing archeological data, largely from the Gobleki Tepe region of the Fertile Crescent, indicate that celiac disease probably emerged as humans transitioned from hunter-gatherer groups to societies dependent on agriculture to secure a stable food supply. Longitudinal studies performed over several decades have suggested that changes in the prevalence of the disease, even apparent epidemic disease, may be due to superimposed or novel environmental factors that may precipitate its appearance. Recent therapeutic approaches are being explored that may supplement, rather than replace, gluten-free diet therapy and permit more nutritional options for future management.

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Section: Emerging Risk Estimatessupporting

confidence: 61%

Celiac Disease: A Disorder Emerging from Antiquity, Its Evolving Classification and Risk, and Potential New Treatment Paradigms

Freeman¹

2015

Gut Liver

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“…Its intake by people suffering from celiac disease causes malabsorption of some important nutrients, such as minerals and folic acid, and mucosal damage in the intestine (1,2). The estimated prevalence of celiac disease worldwide is about 1%, with most cases undiagnosed (3,4). The only effective treatment for celiac disease is to exclude gluten from the diet, throughout the lifespan (2)(3)(4).…”

Section: Introductionmentioning

confidence: 99%

Effects of corn, potato, and tapioca starches on the quality of gluten-free rice bread

Kim

Yun

Jeong

2015

Food Sci Biotechnol

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Corn, potato, and tapioca starches were used to replace proportions of the rice flour in gluten-free breads and their quality characteristics were investigated. The addition of potato starch at 20 or 30% to rice flour improved dough and bread properties such as viscosity parameters, leavening power, uniform air cells, color, and delayed starch retrogradation compared to the rice bread. Moreover, the bread containing 20% potato starch showed the highest score in overall acceptability in sensory evaluation. Overall, the quality of the rice bread was improved by adding starch despite a little positive effect of corn starch.

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“…A case of combined primary hyperparathyroidism, Graves' disease, and celiac disease was reported previously in Lebanon [ 14 ]. There was only one previous domestic case report of celiac disease, but with no coexisting autoimmune diseases [ 15 ]. A few cases in Europe and Canada include adult patients with Graves' disease diagnosed with celiac disease due to chronic diarrhea, malabsorption, or bone deformities [ 16 , 17 , 18 ].…”

Section: Discussionmentioning

confidence: 99%

Celiac Disease in a Predisposed Subject (HLA-DQ2.5) with Coexisting Graves' Disease

et al. 2015

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Celiac disease is an intestinal autoimmune disorder, triggered by ingestion of a gluten-containing diet in genetically susceptible individuals. The genetic predisposition is related to human leukocyte antigen (HLA) class II genes, especially HLA-DQ2-positive patients. The prevalence of celiac disease has been estimated to be ~1% in Europe and the USA, but it is rarer and/or underdiagnosed in Asia. We report a case of celiac disease in a predisposed patient, with a HLA-DQ2 heterodimer, and Graves' disease that was treated successfully with a gluten-free diet. A 47-year-old woman complained of persistent chronic diarrhea and weight loss over a 9 month period. Results of all serological tests and stool exams were negative. However, the patient was found to carry the HLA DQ2 heterodimer. Symptoms improved after a gluten-free diet was initiated. The patient has been followed and has suffered no recurrence of symptoms while on the gluten-free diet. An overall diagnosis of celiac disease was made in a genetically predisposed patient (HLA-DQ2 heterodimer) with Graves' disease.

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A Case of Celiac Disease

Cited by 16 publications

References 8 publications

Celiac Disease: A Disorder Emerging from Antiquity, Its Evolving Classification and Risk, and Potential New Treatment Paradigms

Celiac Disease: A Disorder Emerging from Antiquity, Its Evolving Classification and Risk, and Potential New Treatment Paradigms

Effects of corn, potato, and tapioca starches on the quality of gluten-free rice bread

Celiac Disease in a Predisposed Subject (HLA-DQ2.5) with Coexisting Graves' Disease

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