Ezrin as a complementary marker in ocular toxicity assessment using a three-dimensional reconstructed human corneal-like epithelium model, EpiOcular™

Autophagic tumor-associated macrophages promote the endothelial mesenchymal transition in lung adenocarcinomas through the FUT4/p-ezrin pathway

“…Furthermore, ezrin deletion in autophagic TAMs decreased TGF-β1 expression and contributed to EMT as well as the cell migration and invasion ability of lung adenocarcinomas. Ezrin, an important regulatory protein that connects cell membrane molecules, is highly expressed in various solid tumors and is associated with poor prognosis (35,36). Chen et al reported that ezrin can mediate EMT in A549 cells through the TGF-β1 pathway (37).…”