1998 Basmajian Student Award Paper

Rudolph, Katherine S.; Eastlack, Marty; Axe, Michael J.; Snyder‐Mackler, Lynn

doi:10.1016/s1050-6411(97)00042-4

Cited by 208 publications

(91 citation statements)

References 23 publications

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“…The exact muscle forces are unknown and a limited number of muscles can only be simulated; therefore it seems prob-lematic to transfer these data onto the situation in the living. In vivo studies on femoro-tibia1 translation, on the other hand, have mainly been performed during muscular relaxation [ 19, 34,39], not considering the effect of muscular activation on femoro-tibia1 kinematics, except the work of Beynnon et al [ 3 ] .…”

Section: Introductionmentioning

confidence: 99%

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Femoro‐tibial and menisco‐tibial translation patterns in patients with unilateral anterior cruciate ligament deficiency—a potential cause of secondary meniscal tears

Eisenhart-Rothe

Bringmann

Siebert

et al. 2004

Journal Orthopaedic Research

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Objectiur: To analyze menisco-tibia1 and femoro-tibia1 translation patterns in healthy and ACL-deficient knees in different knee flexion angles under muscle activity.Methods: The ACL-deficient and contralateral healthy knees of 10 patients were examined with an open MR system at 30" and 90" of knee flexion, under isometric contraction of the extensors or flexor muscle groups. Translations between the tibia, the femoral condyles and the menisci were analyzed by three-dimensional image postprocessing.Results: Posterior translation of the femur and menisci relative to the tibia occurred during knee flexion (30-90") in all knees. In ACL-deficient knees, posterior translation of the medial femoral condyle (+I .3 * 3.8 mm) was significantly larger than in healthy knee (-0.9 k 2.9 mm; p < 0.05), while the translation pattern of the menisci was similar (med. meniscus 0.6 f 2.3 mm vs. 0.6 2 2.7 mm). Under isometric contraction of the extensors (relative to the flexor muscle group), an increased posterior position of the femur and menisci was observed at 30" knee flexion, but not at 90". This applied to ACL-deficient and healthy knees.Conclusions: This study shows a significant increase of translation of the medial femoral condyle in ACL-deficient knees, whereas menisco-tibia1 translation remains almost unchanged. This difference in translation patterns indicates that the posterior horn of the medial meniscus might encounter shear, potentially explaining the high rate of secondary medial meniscal tears in patients with ACL-deficiency.

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Section: Introductionmentioning

confidence: 99%

“…It is widely assumed that degenerative changes of the menisci and the cartilage in ACL-deficient patients are caused by excessive displacement between the tibia, femur, and menisci [12,33,34]. Therefore, quantitative data on the relative motion between these structures are required.…”

Section: Introductionmentioning

confidence: 99%

Femoro‐tibial and menisco‐tibial translation patterns in patients with unilateral anterior cruciate ligament deficiency—a potential cause of secondary meniscal tears

Eisenhart-Rothe

Bringmann

Siebert

et al. 2004

Journal Orthopaedic Research

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“…Deficits in the neuromuscular control of both lower extremities following ACLR have been directly implicated in the risk for second ACL injury 126 and may not only be a result of the initial knee injury and subsequent surgery, 23,131,132 but may also characterize the athlete’s preinjury movement patterns. 61,161,162 Therefore, identification and subsequent targeted treatment of aberrant post-ACLR movement patterns are critical not only to maximize functional recovery but also to reduce the risk for a second ACL injury.…”

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confidence: 99%

Neuromuscular Training to Target Deficits Associated With Second Anterior Cruciate Ligament Injury

Stasi¹,

Myer²,

Hewett

2013

Journal of Orthopaedic & Sports Physical Therapy

143

144

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Synopsis Successful return to previous level of activity following anterior cruciate ligament (ACL) reconstruction is not guaranteed, and the prevalence of second ACL injury may be as high as 30%. In particular, younger athletes who return to sports activities within the first several months after ACL reconstruction may be at significantly greater risk of a second ACL rupture compared to older, less active individuals. Significant neuromuscular deficits and functional limitations are commonly identified in athletes following ACL reconstruction, and these abnormal movement and neuromuscular control profiles may be both residual of deficits existing prior to the initial injury and exacerbated by the injury and subsequent ACL reconstruction surgery. Following ACL reconstruction, neuromuscular deficits are present in both the surgical and nonsurgical limbs, and accurately predict second-ACL injury risk in adolescent athletes. While second ACL injury in highly active individuals may be predicated on a number of modifiable and nonmodifiable factors, clinicians have the greatest potential to address the modifiable postsurgical risk factors through targeted neuromuscular interventions. This manuscript will (1) summarize the neuromuscular deficits commonly identified at medical discharge to return to sport, (2) provide the evidence underlying second-ACL injury risk factors, (3) propose a method to assess the modifiable deficits related to second-ACL injury risk, and (4) outline a method of intervention to prevent second ACL injury. The program described in this clinical commentary was developed with consideration for the modifiable factors related to second-injury risk, the principles of motor learning, and careful selection of the exercises that may most effectively modify aberrant neuromuscular patterns. Future validation of this evidence-based, late-phase rehabilitation program may be a critical factor in maximizing return-to-activity success and reduction of second-injury risk in highly active individuals. Level of Evidence Therapy, level 5. J Orthop Sports Phys Ther 2013;43(11):777–792. Epub 10 October 2013. doi:10.2519/jospt.2013.4693

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“…LEWEK et al 2002;RUDOLPH, AXE & SNYDER-MACKLER, 2002;RUDOLPH et al, 1998;SNYDER-MACKLER, DELITO & STRALKA, 1995).…”

Section: Lista De Tabelasmentioning

confidence: 99%

“…Estudos descrevendo a marcha de pacientes com lesão do LCA, sem intervenção cirúrgica, permitem inferir que quando os pacientes se submetem à cirurgia podem já apresentar alterações nos padrões da marcha (DEVITA et al, 1997;LEWEK et al, 2002;RUDOLPH, AXE & SNYDER-MACKLER, 2002;RUDOLPH et al, 1998 A marcha alterada pode trazer conseqüências importantes às estruturas articulares e até musculares, ainda mais ao pensar na freqüência com que este tipo de movimento é realizado no dia a dia. Caso um dos protocolos, CCA ou CCF, recupere de forma mais adequada os padrões da marcha, pode ser esta a melhor opção de trabalho na reabilitação de cirurgia de reconstrução do LCA.…”

Section: Introductionunclassified

Comparação entre dois protocolos de reabilitação após reconstrução do ligamento cruzado anterior através de análise biomecânica

Lima¹

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1998 Basmajian Student Award Paper

Cited by 208 publications

References 23 publications

Femoro‐tibial and menisco‐tibial translation patterns in patients with unilateral anterior cruciate ligament deficiency—a potential cause of secondary meniscal tears

Femoro‐tibial and menisco‐tibial translation patterns in patients with unilateral anterior cruciate ligament deficiency—a potential cause of secondary meniscal tears

Neuromuscular Training to Target Deficits Associated With Second Anterior Cruciate Ligament Injury

Comparação entre dois protocolos de reabilitação após reconstrução do ligamento cruzado anterior através de análise biomecânica

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