“…Another potential mediator is cholecystokinin (CCK), as stimulus originates in the proximal small intestine and is independent of cephalic and gastric phases of diges-meal-stimulated colonic absorption is inhibited following intravenous administration of L-364,718, a specific tion [1]. The enteric nervous system [9], endogenous opiates [10], and a variety of hormones [11,12] have CCK receptor antagonist [13,14].…”