Internal carotid artery aneurysm mimicking normal-tension glaucoma

Netto, Mário Pincelli; Ferrari, Pedro Vanalle; Herrerias, Bruno Torres; Hirai, Flávio E.; Gracitelli, Carolina P. B.

doi:10.5935/0004-2749.20180032

Cited by 3 publications

(2 citation statements)

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“…The assessment of factors including age (less than 50 years old), complaints such as headache or pain around the eye, visual field defects involving the vertical midline, rapid decline in visual acuity, disproportionate neuroretinal rim pallor relative to disc cupping, asymmetric visual acuity and/or visual field loss, and asymmetric cupping can be useful in the differential diagnosis. 7 These factors may increase the likelihood of detecting an intracranial compressive lesion.…”

Section: Discussionmentioning

confidence: 99%

Sphenoid Bone Dysplasia: A Rare Cause of Compressive Optic Neuropathy Mimicking Glaucoma

Kıyat¹,

Karti²,

Esen³

et al. 2023

tjo

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Section: Discussionmentioning

confidence: 99%

Sphenoid Bone Dysplasia: A Rare Cause of Compressive Optic Neuropathy Mimicking Glaucoma

Kıyat¹,

Karti²,

Esen³

et al. 2023

tjo

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“…Compression of the optic nerves by tumors, aneurysms, or normal blood vessels has been reported to cause an optic neuropathy that resembles glaucoma (9–12). In this case series, we describe 3 patients who carried a diagnosis of NTG that had unequivocal and significant radiological compression of the affected optic nerve by a tortuous or dolichoectatic ICA.…”

Section: Discussionmentioning

confidence: 99%

Optic Disc Cupping Due to Dolichoectatic Internal Carotid Artery Optic Nerve Compression

Micieli¹,

Margolin²

2020

Journal of Neuro-Ophthalmology

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Background: To determine whether significant compression of the optic nerve by the internal carotid artery (ICA) can produce an optic neuropathy with optic disc cupping that resembles glaucoma in patients without elevated intraocular pressure (IOP). Methods: This was a retrospective case series of patients referred to neuro-ophthalmology for a possible nonglaucomatous optic neuropathy. Patients were included in the study if they had preserved visual acuity, optic discrelated visual field defects, optic nerve cupping, IOP less than 21 mm Hg, open angles, and unequivocal radiological compression of the ipsilateral optic nerve by an intracranial blood vessel. Results: Three patients were included with a mean age of 56.3 (range 29-82) years. Patient 1 was a 58-year-old man incidentally noted to have left optic nerve cupping on a routine examination. He had an inferior arcuate defect and the left prechiasmatic optic nerve was elevated and compressed by a tortuous left ICA. Patient 2 was a 29-year-old man with a normal-tension glaucoma (NTG) diagnosis for 7 years in the right eye treated with latanoprost. He had a superior greater than inferior arcuate defect and there was vascular compression of the optic nerve between the supraclinoid ICA and A1 segment of the anterior cerebral artery. Patient 3 was an 82-year-old woman with an NTG diagnosis for 10 years who had progression of her visual field defects despite low IOPs. MRI showed mass effect on the right optic nerve by a dolichoectatic right supraclinoid ICA. Conclusions: Significant compression of the optic nerve by a normal, tortuous, or dolichoectatic ICA may result in an optic neuropathy with optic disc cupping that resembles glaucoma.

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Neuro-Ophthalmological Optic Nerve Cupping: An Overview

Waisberg¹,

Micieli²

2021

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Optic nerve cupping or enlargement of the cup-to-disc ratio is widely recognized as a feature of glaucoma, however it may also occur in non-glaucomatous optic neuropathies. The most well-recognized non-glaucomatous optic neuropathies that cause cupping include compressive optic neuropathies, arteritic anterior ischemic optic neuropathies, hereditary optic neuropathies, and optic neuritis. Cupping is thought to consist of two main components: prelaminar and laminar thinning. The former is a shallow form of cupping and related to loss of retinal ganglion cells, whereas the latter involves damage to the lamina cribrosa and peripapillary scleral connective tissue. Differentiating glaucomatous and non-glaucomatous optic nerve cupping remains challenging even for experienced observers. Classically, the optic nerve in non-glaucomatous causes has pallor of the neuroretinal rim, but the optic nerve should not be examined in isolation. The patient’s medical history, history of presenting illness, visual function (visual acuity, color vision and visual field testing) and ocular examination also need to be considered. Ancillary testing such as optical coherence tomography of the retinal nerve fiber layer and ganglion cell layer-inner plexiform layer may also be helpful in localizing the disease. In this review, we review the non-glaucomatous causes of cupping and provide an approach to evaluating a patient that presents with an enlarged cup-to-disc ratio.

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Internal carotid artery aneurysm mimicking normal-tension glaucoma

Cited by 3 publications

References 0 publications

Sphenoid Bone Dysplasia: A Rare Cause of Compressive Optic Neuropathy Mimicking Glaucoma

Sphenoid Bone Dysplasia: A Rare Cause of Compressive Optic Neuropathy Mimicking Glaucoma

Optic Disc Cupping Due to Dolichoectatic Internal Carotid Artery Optic Nerve Compression

Neuro-Ophthalmological Optic Nerve Cupping: An Overview

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