COVID-19 myocarditis: a case report

Abstract

A male patient with flu-like symptoms and tomography and laboratory diagnosis of severe acute respiratory syndrome. He developed acute cardiac dysfunction during admission and was submitted to a cardiac magnetic resonance imaging examination, which confirmed acute myocarditis, indicating cardiac involvement by coronavirus disease 2019. A review and discussion about coronavirus disease 2019-related cardiac manifestations are reported, focusing on the imaging findings to make diagnosis.

Acute myocarditis associated with secondary cerebral edema in a patient with COVID-19: Case report

“…(3) In addition to causing damage to the respiratory system, SARS-CoV-2 can trigger cardiac and brain injury. (4) In some cases of myocardial injury, SARS-CoV-2 particles have been identi ed in the myocardium, reinforcing the hypothesis that cardiotoxicity occurs. (5) Likewise, studies suggest that the virus can infect the brain directly, reaching its parenchyma through the bloodstream and the olfactory system.…”

“…However, gas exchange impaired by continuous blood ow to lung regions of low ventilation causes a ventilation-perfusion mismatch, resulting in blood hypoxemia and, consequently, in worsening of oxygenation of the cardiac tissue. (11) Yokoo et al (4) described the case of a patient diagnosed with COVID-19, who was over 80 years old and had a history of hypertension and ischemic stroke. Laboratory tests showed high levels of troponin T, electrocardiogram showed no signs of ischemia, and echocardiogram showed an ejection fraction of 35%.…”

Acute myocarditis associated with secondary cerebral edema in a patient with Covid-19: Case report

“…However, gas exchange impaired by continuous blood ow to lung regions of low ventilation causes a ventilationperfusion mismatch resulting in blood hypoxemia, and consequently in worsening oxygenation of cardiac tissue. (19) Yokoo et al, (7) presented the case of a patient over 80 years old with a history of hypertension and ischemic stroke. Laboratory tests showed high levels of troponin T, electrocardiogram showed no signs of ischemia, and echocardiogram showed an ejection fraction of 35%.…”

“…This patient underwent a cardiovascular MRI that revealed areas of delayed enhancement with ischemia in the left ventricular base septal wall, in addition to diffuse hypokinesia and impaired global systolic function. (7) Cases of myocarditis associated with COVID-19 have been reported from young to old. Paul et al, (20) described the case of a 35-year-old patient who was overweight as the only cardiovascular risk.…”

“…(5,6) In addition to causing damage to the respiratory system, SARS-CoV-2 can trigger cardiac and brain injury. (7,8) The mechanisms of damage to these two systems are not fully understood and are probably multifactorial. (6) In some cases of myocardial injury, SARS-CoV-2 particles have been identi ed in the myocardium, reinforcing the hypothesis that cardiotoxicity occurs.…”