A Case of Pseudo-Hypertrophic Cardiomyopathy: A Congenital Heart Disease

Ieva, Riccardo; Correale, Michele

doi:10.1590/s1807-59322008000400026

Cited by 2 publications

(5 citation statements)

References 7 publications

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“… 1 It is important to remember that the pathogenesis of LVH includes angiotensin II receptors and membrane-stretched cardiomyocytes. 2 …”

Section: Discussionmentioning

confidence: 99%

“…The high prevalence of ventricular dysfunction is used to support the view that the ability to regenerate after injury is not significant. However, research has shown that myocyte regeneration is fundamental to heart homeostasis 2. For instance, during aortic stenosis, the combination of hypertrophy and hyperplasia increases cardiac mass3 at specific locations, such as the site of a therapeutic stem cell implant 2…”

Section: Introductionmentioning

confidence: 99%

“…However, research has shown that myocyte regeneration is fundamental to heart homeostasis. 2 For instance, during aortic stenosis, the combination of hypertrophy and hyperplasia increases cardiac mass 3 at specific locations, such as the site of a therapeutic stem cell implant. 2 …”

Section: Introductionmentioning

confidence: 99%

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Anti-Hypertensive Drugs Have Different Effects on Ventricular Hypertrophy Regression

Ferreira

Abreu

Valenti

et al. 2010

Clinics

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OBJECTIVES:There is a direct relationship between the regression of left ventricular hypertrophy (LVH) and a decreased risk of mortality. This investigation aimed to describe the effects of anti-hypertensive drugs on cardiac hypertrophy through a meta-analysis of the literature.METHODS:The Medline (via PubMed), Lilacs and Scielo databases were searched using the subject keywords cardiac hypertrophy, antihypertensive and mortality. We aimed to analyze the effect of anti-hypertensive drugs on ventricle hypertrophy.RESULTS:The main drugs we described were enalapril, verapamil, nifedipine, indapamina, losartan, angiotensin-converting enzyme inhibitors and atenolol. These drugs are usually used in follow up programs, however, the studies we investigated used different protocols. Enalapril (angiotensin-converting enzyme inhibitor) and verapamil (Ca++ channel blocker) caused hypertrophy to regress in LVH rats. The effects of enalapril and nifedipine (Ca++ channel blocker) were similar. Indapamina (diuretic) had a stronger effect than enalapril, and losartan (angiotensin II receptor type 1 (AT1) receptor antagonist) produced better results than atenolol (selective β1 receptor antagonist) with respect to LVH regression.CONCLUSION:The anti-hypertensive drugs induced various degrees of hypertrophic regression.

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“… 1 It is important to remember that the pathogenesis of LVH includes angiotensin II receptors and membrane-stretched cardiomyocytes. 2 …”

Section: Discussionmentioning

confidence: 99%

Section: Introductionmentioning

confidence: 99%

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Anti-Hypertensive Drugs Have Different Effects on Ventricular Hypertrophy Regression

Ferreira

Abreu

Valenti

et al. 2010

Clinics

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“…They attained diagnosis was made by echocardiogram but no CMR or histopathological study was performed. 5 In our case, CMR was performed, and showed symmetric hypertrophy of the SRV with areas of fibrosis. Also, the global longitudinal strain reported an abnormal value (−16%), which suggests the alteration of the myofibril, however these parameters have not been evaluated for a SRV.…”

Section: Discussionmentioning

confidence: 56%

“…Leva et al., had already described a case of pseudo‐HCM in a 65‐year‐old patient without obstruction in the outflow tract of the SRV that would explain the hypertrophy. They attained diagnosis was made by echocardiogram but no CMR or histopathological study was performed 5 . In our case, CMR was performed, and showed symmetric hypertrophy of the SRV with areas of fibrosis.…”

Section: Discussionmentioning

confidence: 70%

Hypertrophic cardiomyopathy in the systemic right ventricle in a patient with congenitally corrected transposition of the great arteries: A case report

García‐Cruz,

Manzur‐Sandoval,

Toledo‐Alemán

et al. 2023

Echocardiography

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Congenitally corrected transposition of the great arteries is a rare clinical entity, which usually presents during adulthood with associated defects; atrioventricular block, heart failure, systemic valve failure, and arrhythmias usually complicate the clinical course. Even rarer is associated hypertrophic cardiomyopathy, which complicates the disease course and clinical decision‐making. Herein, we present a patient with this condition who underwent heart transplantation, with adequate clinical resolution.

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A Case of Pseudo-Hypertrophic Cardiomyopathy: A Congenital Heart Disease

Cited by 2 publications

References 7 publications

Anti-Hypertensive Drugs Have Different Effects on Ventricular Hypertrophy Regression

Anti-Hypertensive Drugs Have Different Effects on Ventricular Hypertrophy Regression

Hypertrophic cardiomyopathy in the systemic right ventricle in a patient with congenitally corrected transposition of the great arteries: A case report

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