Hiperfosfatasemia transitória benigna da infância

Alves, Crésio de Aragão Dantas; Arruti, Renata

doi:10.1590/s1413-78522009000100011

Cited by 3 publications

(1 citation statement)

References 10 publications

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“…Twenty‐six pertinent reports were found in the references of the mentioned reports. Hence, a total of 142 reports were included in the final analysis (9–150) (references 51–150 are online only: http://links.lww.com/MPG/A215): 84 in English (9–92), 8 in French (93–100), 5 in German (101–105), 18 in Italian (106–123), 1 in Portuguese (124), and 26 in Spanish (125–150). They had been reported from Italy (N = 22), Spain (N = 21), the United States (N = 19), Great Britain (N = 14), Germany (N = 11), France (N = 9), Czech Republic (N = 7), Argentine (N = 6), Belgium (N = 3), Canada (N = 3), Japan (N = 3), Australia (N = 2), Brazil (N = 2), Croatia (N = 2), Denmark (N = 2), Israel (N = 2), Ireland (N = 2), Scotland (N = 2), Switzerland (N = 2), Venezuela (N = 2), Finland (N = 1), Greece (N = 1), China (N = 1), Poland (N = 1), Sweden (N = 1), and Turkey (N = 1).…”

Section: Resultsmentioning

confidence: 99%

Transient Benign Hyperphophatasemia

Gualco

Lava

Garzoni

et al. 2013

J. pediatr. gastroenterol. nutr.

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Transient benign hyperphosphatasemia is likely the most common cause of hyperphosphatasemia among healthy infants and toddlers. Sometimes it also occurs in older children and adults, indicating that the traditional term transient benign hyperphosphatasemia of infancy and early childhood may not be correct. The elevation in alkaline phosphatase persists for >4 months in ≈20% of the cases. Recognition of this benign condition is crucial to avoid unnecessary investigations.

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Section: Resultsmentioning

confidence: 99%

Transient Benign Hyperphophatasemia

Gualco

Lava

Garzoni

et al. 2013

J. pediatr. gastroenterol. nutr.

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Effects of Tin on Enzyme Activity in Holothuria grisea (Echinodermata: Holothuroidea)

Pereira

Mattar

Pereira

et al. 2016

Bull Environ Contam Toxicol

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This study evaluated the effect of tin exposure on enzyme activity in the sea cucumber (Holothuria grisea Selenka, 1867). After exposure to 0 (control), 0.04, 0.08, or 0.12 mg L tin, we tested the activities of total cholinesterase in longitudinal muscles, acid phosphatase in gonads and the respiratory tree, as well as alkaline phosphatase in the intestines during a 96-h bioassay. Regression analyses showed that all enzyme activities declined with increasing tin concentrations, except for acid phosphatase in the respiratory tree, which were similarly, inhibited at all tin concentrations. These results indicate that H. grisea is a potential bioindicator for seascape habitat monitoring programs, as its biochemical markers show sensitivity to trace elements that can indicate a rise in pollution levels.

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Abrin Poisoning in an 18-Month-Old Child

2015

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Patient: Male, 1Final Diagnosis: Abrin poisoningSymptoms: Abdominal pain • diarrhea • vomitingMedication: —Clinical Procedure: —Specialty: Pediatrics and NeonatologyObjective:Rare diseaseBackground:When discussing known poisons and their clinical consequences, few physicians are aware of the deadly poison abrin. The common symptoms of abrin toxicity include nausea, vomiting, abdominal pain, and diarrhea with or without bloody stool. However, with an estimated fatal human dose of less than 1 microgram/kg, death due to complications such as liver failure, renal failure, and cerebral edema are possible.Case Reports:An 18-month-old male presented to the emergency department with an abrupt onset of fever, vomiting, diarrhea, and dehydration. The parents had assumed the child was suffering from a severe gastroenteritis until they noticed 3 consecutive diapers containing colorful seeds that were identified by poison control as belonging to the Abrus precatorius plant. The child’s gastrointestinal symptoms were consistent with reported cases of abrin poisoning, but the patient also had an isolated and significantly elevated alkaline phosphatase, with testing unable to reveal the responsible pathological process.Conclusions:To reduce morbidity and possible mortality, parents and pediatricians alike should be conscious of the danger the Abrus precatorius seed poses to the pediatric population. Children are at a greater risk of ingesting these seeds due to their colorful appearance, and the consequences could be fatal. Through this case report we hope to raise public awareness regarding this toxin. This includes the management of known cases, as well as the possibility of encountering an isolated elevated alkaline phosphatase level as a laboratory finding if ingestion occurs.

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Hiperfosfatasemia transitória benigna da infância

Cited by 3 publications

References 10 publications

Transient Benign Hyperphophatasemia

Transient Benign Hyperphophatasemia

Effects of Tin on Enzyme Activity in Holothuria grisea (Echinodermata: Holothuroidea)

Abrin Poisoning in an 18-Month-Old Child

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