“…different hormone secreting (prolactin [PRL]-, growth hormone [GH]-, PRL-GH-, thyroid-stimulating hormone [TSH]-, and adrenocorticotropic hormone [ACTH]) pituitary adenomas , precocious puberty, adrenal primary hyperplasia, hypophosphatemic rickets/osteomalacia, and even ovarian cysts. 2,6,11,[14][15][16] Secreting üituitary adenomas are reported in association with more than 50% of the MAS cases. 14 As well as endocrinologic disorders, fibrous dysplastic bone changes in MAS occur in various forms, such as expansions, cystic lesions, and fibrous thickening that, quite unlike the sclerotic tissues, have an active vascular network in the interior.…”