Pseudotumor cerebral sem edema de papila óptica

Jc, Seggia; Ml, De Menezes

doi:10.1590/s0004-282x1993000400015

Cited by 3 publications

(1 citation statement)

References 41 publications

Supporting

Mentioning

Contrasting

Order By: Relevance

“…Years ago, Ossoinig suggested the use of the stretch-test (originally called the "30 degrees-test"): in widened optic nerve patterns due to fluid around the optic nerve parenchyma, a decreased optic nerve thickness was observed after performing the stretch-test (positive test result), whereas in solid lesions of the optic nerve no change of optic nerve thickness was found (negative test result) (Haritoglou et al 2002). In patients with increased ICP due to any cause, either ophthalmoscopic evaluation or bilateral retrobulbar ultrasound is mandatory, as asymmetric and unilateral papilledemae in patients with IIH are well described (Seggia and De Menezes 1993). In addition, Bäuerle et al did not find any correlation of papilledema and OND with CSF reduction in the short-term follow-up.…”

Section: Assessment Of Intracranial Pressurementioning

confidence: 99%

Application of Orbital Sonography in Neurology

Ertl¹,

Gamulescu²,

Schlachetzki³

2012

Sonography

View full text Add to dashboard Cite

Section: Assessment Of Intracranial Pressurementioning

confidence: 99%

Application of Orbital Sonography in Neurology

Ertl¹,

Gamulescu²,

Schlachetzki³

2012

Sonography

View full text Add to dashboard Cite

Cerebrospinal fluid dynamics between the intracranial and the subarachnoid space of the optic nerve. Is it always bidirectional?

Killer

Jaggi²,

Flammer

et al. 2007

Brain

225

180

View full text Add to dashboard Cite

CSF is thought to flow continuously from the site of production in the ventricles into interconnected spaces; i.e. cisterns and subarachnoid spaces (SASs). Since the SAS of the optic nerve is defined by a cul-de-sac anatomy, it is not evident how local CSF might recycle from that region to the general SAS. The concept of free communication of CSF has recently been challenged by the description of a concentration gradient of beta-trace protein, a lipocalin-like prostaglandin d-synthase (L-PGDS), between the spinal CSF and that in the SAS of the optic nerve, indicating diminished local clearance or local overproduction of L-PGDS here. In fact, computed cisternography with a contrast agent in three patients with idiopathic intracranial hypertension and asymmetric papilloedema demonstrate a lack of contrast-loaded CSF in the SAS of the optic nerve despite it being present in the intracranial SAS, thus suggesting compartmentation of the SAS of the optic nerve. The concept of an optic nerve compartment syndrome is further supported by a concentration gradient of brain-derived L-PGDS between the spinal CSF and the CSF from the optic nerve SAS in the same patients.

show abstract

A Comparison of Idiopathic Intracranial Hypertension With and Without Papilledema

et al. 2009

View full text Add to dashboard Cite

Objective To compare clinical features, visual characteristics, and treatment of idiopathic intracranial hypertension patients with and without papilledema. Background Idiopathic intracranial hypertension does not often occur without papilledema. This study estimates the prevalence and compares the clinical characteristics of idiopathic intracranial hypertension patients with and without papilledema. Methods We performed a cross-sectional analysis of all idiopathic intracranial hypertension patients diagnosed at the University of Utah Neuro-Ophthalmology Unit between 1990 and 2003. Patient records were reviewed for presence of papilledema and other signs, symptoms, and treatment characteristics. Each patient without papilledema was matched to the patient with papilledema who was closest to his/her age and sex. McNemar’s and Wilcoxon-signed rank sum tests were used to compare characteristics between matched pairs. Results Among all patients (n = 353), the prevalence of those without papilledema was 5.7% (n = 20). Patients without papilledema reported photopsias (20%), and were found to have spontaneous venous pulsations (75%) and non-physiologic visual field constriction (20%) more often than did those with papilledema. Mean opening pressure, although above normal, was lower in patients without papilledema (mean = 309 mm cerebrospinal fluid) compared with those with papilledema (mean = 373 mm cerebrospinal fluid, P = .031). Idiopathic intracranial hypertension patients without papilledema had more frequent diagnostic lumbar punctures than did patients with papilledema. Visual acuities and treatment were similar between groups. Conclusions The clinical presentation of idiopathic intracranial hypertension without papilledema is only somewhat different from that of idiopathic intracranial hypertension with papilledema. The lower opening pressure in patients without papilledema may explain variations in symptoms and signs between the 2 groups. When there are visual field changes in idiopathic intracranial hypertension without papilledema, non-physiologic visual loss should be considered.

show abstract

Pseudotumor cerebral sem edema de papila óptica

Cited by 3 publications

References 41 publications

Application of Orbital Sonography in Neurology

Application of Orbital Sonography in Neurology

Cerebrospinal fluid dynamics between the intracranial and the subarachnoid space of the optic nerve. Is it always bidirectional?

A Comparison of Idiopathic Intracranial Hypertension With and Without Papilledema

Contact Info

Product

Resources

About