Crises focais na hiperglicemia nao cetótica

Guerreiro, Carlos A. M.; Falcão, Antonio E.; Silveira, Diosely C.

doi:10.1590/s0004-282x1991000400014

Cited by 4 publications

(1 citation statement)

References 6 publications

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“…Epileptic seizures in diabetic patients are mostly reported as focal seizures (13,14,15,16,17,18,19) with or without secondary generalization; epilepsy partialis continua (EPC) (20,21,22,23,24,25,26,27,28), occipital seizures (29,30,31,32,33), and reflex seizures (34,35,36) have also been described. However, some cases of generalized epilepsy (childhood absence epilepsy, juvenile absence epilepsy, tonic-clonic seizures on wakening, and juvenile myoclonic epilepsy) associated with T1DM have also been reported (7,9,37,38).…”

Section: Introductionmentioning

confidence: 99%

MECHANISMS IN ENDOCRINOLOGY: Seizures and type 1 diabetes mellitus: current state of knowledge

Verrotti

Scaparrotta

Olivieri

et al. 2012

European Journal of Endocrinology

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In this review, we will try to analyze the possible coexistence between epilepsy or seizures and type 1 diabetes mellitus (T1DM), in order to establish if there is more than a casual association, and to investigate possible mechanisms underlying this link. Anti-glutamic acid decarboxylase antibodies (GAD-Abs) have been associated with T1DM and a great number of neurological diseases such as epilepsy. Epilepsy can be a feature of a large variety of autoimmune or inflammatory disorders. GAD-Abs can have a role at the basis of the possible link between epilepsy and T1DM, although their real pathogenetic mechanism in neurological diseases is still unknown. Metabolic conditions such as hypoglycemia and hyperglycemia, common problems in diabetic patients, may be also implicated, even if their underlying mechanism is minimally understood.

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