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Abstract

75Arq Bras Endocrinol Metab. 2012;56/1 Table 1. Baseline characteristics of patients initially randomized to the intervention groups and patients that concluded the study, according to the intervention group (placebo or L-T4 use) Patients initially randomized* Patients that concluded the study* L-T4 (n = 17) Placebo (n = 16) L-T4 (n = 9) Placebo (n = 13)Age (years) 49.9 ± 9.3 (49) 44.6 ± 8.1 (45) 51.7 ± 10.2 (49) 44.4 ± 8.9 (44) TSH (µUI/mL) 7.6 ± 3.6 (7.2) 7.9 ± 3.2 (6.8) 8.6 ± 3.1 (8.4) 8.2 ± 3.4 (7.8) Conclusions: Our findings suggest that blood pressure levels in patients with active acromegaly are very similar, and depend on excess GH. However, once the disease becomes controlled and IGF-1 levels decrease, their blood pressure levels will depend on the other cardiovascular risk factors. Leia-se:Conclusions: Blood pressure (BP) levels in patients with active acromegaly dependent of the GH excess. However, once the disease becomes controlled and IGF-1 levels decrease, their blood pressure levels are depend on the other cardiovascular risk factors. Villarroel and cols. (40), from our group, reported that girls with PCOM have higher AMH levels than girls without PCOM (72.5 ± 6.1 vs. 33.4 ± 2.6 pmol/L; P < 0.0001), and lower FSH levels (5.4 ± 0.3; 6.2 ± 0.2 mUI/ml; P < 0.036). Leia-se:Villarroel and cols. (40), from our group, reported that girls with PCOM have higher AMH levels than girls without PCOM (72.5 ± 6.1 vs. 33.4 ± 2.6 pmol/L; P < 0.0001), and lower FSH levels (5.4 ± 0.3 vs. 6.2 ± 0.2 mUI/ml; P < 0.036). Na página 595, segunda coluna, último parágrafo onde se lê:In 160 obese adolescents girls, the subgroup with the metabolic syndrome (48 girls) demonstrated significantly higher testosterone and DHEA-S concentrations compared with the 112 girls without MS. Leia-se:In 160 obese adolescents girls, the subgroup with the metabolic syndrome (MS) (48 girls) demonstrated significantly higher testosterone and DHEA-S concentrations compared with the 112 girls without MS.Na página 595, na legenda da figura, onde se lê: 79Arq Bras Endocrinol Metab. 2012;56/1 Na página 596, Table 2, onde se lê: Potential phenotypes