Aphonia after shoulder surgery: case report

Moreno, Carlos; Fonseca, Sara

doi:10.1016/j.bjane.2013.09.014

Cited by 1 publication

(2 citation statements)

References 14 publications

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“…If the RLN paralysis or injury is bilateral, then aphonia develops due to paralysis of both vocal folds. 5,6 RLN is the branch of the vagus nerve, and the incidence of such a condition due to vagus nerve paralysis is reported to be 1:200 to 1:1200. 2 The incidence or prevalence of aphonia in patients undergoing surgery with spinal anesthesia is not well known.…”

Section: Discussionmentioning

confidence: 99%

“…Unilateral RLN injury paralyzes the ipsilateral vocal fold, and the clinical symptoms of dysphonia or hoarseness appear. If the RLN paralysis or injury is bilateral, then aphonia develops due to paralysis of both vocal folds 5,6 . RLN is the branch of the vagus nerve, and the incidence of such a condition due to vagus nerve paralysis is reported to be 1:200 to 1:1200 2 …”

Section: Discussionmentioning

confidence: 99%

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Transient aphonia following spinal anesthesia during emergency cesarean section: Case report and review of literature

Adhikari,

Ghimire,

Adhikari

et al. 2023

Clinical Case Reports

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Key Clinical MessageSubarachnoid block with local anesthetic agents is a well‐established anesthesia technique among pregnant females for labor analgesia and cesarian delivery. Although it is considered a reliable and safe technique for both mothers and fetuses, unexpected high or low levels of block may occur due to accidental injection of these agents into different meningeal spaces other than intended. Hypotension, bradycardia, headache, and failed anesthesia are common complications of spinal anesthesia. Though rare, neurological complications like aphonia, dysphagia, and tingling sensation have also been reported. The article reports a case of a 22‐year‐old primigravida who sustained transient aphonia following intrathecal administration of bupivacaine for an emergency cesarian section for meconium‐stained liquor with fetal distress. There were no other neurological manifestations or features suggestive of high spinal block. The sensory level of the block was fixed to T6 with hemodynamic stability throughout aphasia with an episode of hypotension preceding aphonia. Aphonia commenced 9 min after the spinal anesthesia continued for a total duration of 15 min. Neurological examination, relevant investigations, and consultations were done to make a diagnosis. Thus, it is important to be aware of the possible neurological complications associated with spinal anesthesia.

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Section: Discussionmentioning

confidence: 99%

Section: Discussionmentioning

confidence: 99%