1 - Équilibre pelvien et instabilité des prothèses totales de hanche

Eddine, T. Ala

doi:10.1016/s0035-1040(04)70179-8

Cited by 3 publications

(2 citation statements)

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“…Most studies into iliopsoas tendonitis after hip arthroplasty investigate its incidence 6–9,17–19 or the success of different treatment paths, 20–23 which include physical therapy, corticosteroid injections, iliopsoas tenotomy, and acetabular cup revision. Only one previous study has attempted to simulate iliopsoas impingement pre‐ or postoperatively in patients who have undergone THA and been diagnosed with iliopsoas tendonitis.…”

Section: Introductionmentioning

confidence: 99%

“…No studies have done so in a sample of patients who have undergone HRA. However, with an incidence of between 18% and 30% for HRA, compared to 4%–7% for THA, the incidence of groin pain after HRA is meaningfully higher, 6–9,17,18,24,25 and theories have been proposed for causes of groin pain that are independent of impingement with the acetabular cup 12,24,26,27 . Therefore, we sought to examine the mechanism of iliopsoas tendonitis in HRA patients using a case‐controlled investigation of symptomatic and asymptomatic patients, and compare these results to previously collected results of THA patients in a similarly designed study to understand if the mechanisms of cup impingement and tendonitis differ 28 …”

Section: Introductionmentioning

confidence: 99%

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Comparison of iliopsoas tendonitis after hip resurfacing arthroplasty and total hip arthroplasty: A case‐controlled investigation using a validated simulation

Hardwick‐Morris,

Twiggs,

Miles

et al. 2024

Journal Orthopaedic Research

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Iliopsoas tendonitis, typically caused by impingement with the acetabular cup, occurs in up to 18% of patients after total hip arthroplasty (THA) and up to 30% of patients after hip resurfacing arthroplasty (HRA). We have developed a simulation for detecting iliopsoas impingement and validated it in a previous study of THA patients. However, due to the difference in incidence between HRA and THA, this study had two aims. First, to validate the simulation in a cohort of HRA patients and, second, to comparethe results of the HRA and THA patients to understand any differences in their etiology. We conducted a retrospective search in an experienced surgeon's database for HRA patients with iliopsoas tendonitisand control patients without iliopsoas tendonitis, resulting in two cohorts of 12 patients. Using CT scans, 3D models of the each patient's prosthetic and bony anatomy were generated, landmarked, and simulated. Regarding validation of the simulation for HRA patients, impingement significantly predicted the probability of iliopsoas tendonitis in logistic regression models and the simulation had a sensitivity of 83%, specificity of 100%, and an AUC ROC curve of 0.95. Unexpectedly, the HRA cohort exhibited less impingement than the THA cohort. Our novel simulation has now been demonstrated to detect iliopsoas impingement and differentiate between the symptomatic and asymptomatic cohorts in investigations of THA and HRA patients. This tool has the potential to be used preoperatively, to guide decisions about optimal cup placement, and postoperatively, to assist in the diagnosis of iliopsoas tendonitis.

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Section: Introductionmentioning

confidence: 99%