channelopathy ͉ FEB3 locus ͉ convulsions ͉ epilepsy ͉ neuronal excitability I t has long been known that there is a major genetic component in the etiology of febrile seizures (FS), and an autosomaldominant (AD) inheritance with incomplete penetrance has been proposed in large pedigrees or groups of families with FS (1). Six loci for familial FS have been reported, but no genes were identified; they have been mapped at chromosomes 8q13-21
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