Superior semicircular canal dehiscence syndrome (SSCDS) is described presently as a disorder of the inner ear [1], denoted by the absence of a bony covering of this canal [2]. This acts as a "third window" anomaly, modifying the physiological functions of the inner ear, which generate an abnormal transmission of vibration to the vestibular system, responsible for the clinical symptomatology, such as vertigo, nystagmus, oscillopsia, and disequilibrium prompted by loud sounds (Tullio phenomenon) [3,4]. Furthermore, the Valsalva maneuver stimulates shifts in the pressure of the middle or intracranial ear because of pressure changes in the
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