We present the final results of the spin asymmetries A 1 and the spin structure functions g 1 of the proton and the deuteron in the kinematic range 0.0008ϽxϽ0.7 and 0.2ϽQ 2 Ͻ100 GeV 2 . For the determination of A 1 , in addition to the usual method which employs inclusive scattering events and includes a large radiative background at low x, we use a new method which minimizes the radiative background by selecting events with at least one hadron as well as a muon in the final state. We find that this hadron method gives smaller errors for xϽ0.02, so it is combined with the usual method to provide the optimal set of results.

We present a measurement of semi-inclusive spin asymmetries for positively and negatively charged hadrons from deep inelastic scattering of polarised muons on polarised protons and deuterons in the range 0.0031 GeV2. Compared to our previous publication on this subject, with the new data the statistical errors have been reduced by nearly a factor of two. From these asymmetries and our inclusive spin asymmetries we determine the polarised quark distributions of valence quarks and non-strange sea quarks at Q2=10 GeV2. The polarised u valence quark distribution, Δuv(x), is positive and the polarisation increases with x. The polarised d valence quark distribution, Δdv(x), is negative and the non-strange sea distribution, is consistent with zero over the measured range of x

We present a new measurement of the virtual photon proton asymmetry A p 1 from deep inelastic scattering of polarized muons on polarized protons in the kinematic range 0:0008 < x < 0:7 and 0:2 < Q 2 < 100 GeV 2 . With this, the statistical uncertainty of our measurement has improved by a factor of 2 compared to our previous measurements. The spin-dependent structure function g p 1 is determined for the data with Q 2 > 1 GeV 2 . A perturbative QCD evolution in next-to-leading order is used to determine g p 1 (x) at a constant Q 2 . A t Q 2 = 10 GeV 2 we nd, in the measured range, R 0:7 0:003 g p 1 (x)dx = 0 : 139 0:006 (stat) 0:008 (syst) 0:006 (evol).The value of the rst moment p 1 = R 1 0 g p 1 ( x )dx of g p 1 depends on the approach used to describe the behaviour of g p 1 at low x. We nd that the Ellis-Jae sum rule is violated. With our published result for d 1 we conrm the Bjorken sum rule with an accuracy of 15% at the one standard deviation level.

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P Pu ur rp po os se e: : To evaluate the effects of sedation with sufentanil on respiratory drive, respiratory pattern, and gas exchange of critically ill patients during pressure support ventilation. M Me et th ho od ds s: :In this prospective observational cohort study, we observed 12 adult patients receiving partial ventilatory support for acute respiratory failure. Each subject received a continuous infusion of sufentanil at 0.2 to 0.3 µg·kg to obtain a modified Ramsay sedation score between 2 and 3. In basal conditions and at variable distance from the beginning of the sufentanil infusion (10', 30', 60', 120', 24 hr) we evaluated gas exchange, hemodynamic variables, respiratory rate (RR), tidal volume (TV), respiratory pattern, respiratory drive (P0.1) and inspiratory impedance of the respiratory system [P0.1/TV/inspiratory time (Ti)]. of sufentanil resulted in the desired level of sedation. No significant heart rate, heart rhythm and blood pressure changes were observed. Sufentanil infusion did not affect TV, minute volume, Ti/inspiratory duty cycle, RR, P0.1, P0.1/TV/Ti and gas exchange did not change significantly over the study period.C Co on nc cl lu us si io on n: : A continuous infusion of sufentanil induces "awake" sedation with no detectable effects on respiratory variables in critically ill patients during partial ventilatory support. EDATION and analgesia are widely used in intensive care unit (ICU) patients, particularly during mechanical ventilation.Critically ill patients often experience stressful maneuvers such as endotracheal intubation and mechanical ventilation, suctioning or painful diagnostic and therapeutic interventions. The net effect is an increase in catecholamine secretion and oxygen demand with further systemic and coronary vasoconstriction.1 An adequate level of analgesia and sedation (analgesia-sedation) is thus a precious tool to control