Objective. Hyperthyroidism has been described as elevated serum free T3 and/or
free T4 levels with decreased thyrotropin (TSH) concentrations. The main causes are related to
autoimmune and neoplastic pathology. However, it might be caused due to a long-term topical
exposure (iodine solution dressing) or by intravenous administration of
iodine-containing substances.
Both clinical and laboratory features might be presented. The
main management is based on interruption of all exposures with
iodine solutions and also antithyroid medicine in case of severe
laboratory and clinical disturbances.
Data Sources.
We present a case of iodine-induced hyperthyroidism
in a critically ill ICU patient caused by excessive iodine
containing antiseptic solution washes and contrast agent administration.
The patient was successfully treated by discontinuing iodine exposure and
beta-blocker administration.
Conclusions.
In patients with underlying thyroid gland pathology, thyroid-function
tests and clinical observation in the ICU are of critical importance.
Page kidney is a well-known phenomenon causing hypertension, due to compression of renal parenchyma by a subcapsular hematoma, of either traumatic or non-traumatic origin. The main therapeutic approach is based on surgical approach (nephrectomy or hematoma evacuation) and antihypertensive treatment. In this paper we present a post-traumatic case of Page Kidney in a Critical Care unit. We discuss different therapeutical opportunities to extremely elevated systemic blood pressure resistant to traditional drug therapy.
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