Vasculitis IgA como forma de presentación de la infección por el virus de inmunodeficiencia humana

Brandy-García, Anahy M.; Santos‐Juanes, Jorge; Suárez, S.; Caminal-Montero, Luis

doi:10.1016/j.reuma.2018.03.010

Cited by 6 publications

(4 citation statements)

References 6 publications

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“…The treatment of HSP in HIV infected patients is not well codified and it is based on case reports. The use of ART was efficacious leading to disappearance of cutaneous articular and abdominal signs in some reported cases [12,15,16]. Complete remission of our patient after ART can also support the above mentioned pathogenic mechanism of HSP in HIV infection (increased circulation of immune complexes and local replication of HIV).…”

Section: Discussionsupporting

confidence: 78%

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Henoch-schonlein purpura vasculitis revealing human immunodeficiency virus infection.

Bahloul¹,

Frikha²,

Abderrahmen³

et al. 2018

immunology-case-reports.

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Section: Discussionsupporting

confidence: 78%

“…HSP in adults can reveal or be associated with HIV, few cases have been reported [10][11][12][13][14][15][16]. The pathogenesis of HSP in patients with HIV infection remains hypothetic.…”

Section: Discussionmentioning

confidence: 99%

Henoch-schonlein purpura vasculitis revealing human immunodeficiency virus infection.

Bahloul¹,

Frikha²,

Abderrahmen³

et al. 2018

immunology-case-reports.

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“…Consistently, many types of bacteria and viruses are thought to be associated with the pathogenesis of IgA vasculitis. The representative causative pathogens are Streptococcus [ 22 , 23 , 24 ], S. aureus [ 25 , 26 , 27 , 28 ], Helicobacter pylori [ 29 , 30 ], varicella-zoster virus [ 31 , 32 ], hepatitis virus [ 33 ], Parvovirus [ 34 , 35 ], human immunodeficiency virus [ 36 ], cytomegalovirus [ 37 ], and Clostridium difficile [ 38 ]. Therefore, these microorganisms are triggers for the development of IgA vasculitis.…”

Section: Triggers and Pathogenesis Of Iga Vasculitismentioning

confidence: 99%

IgA Vasculitis: Etiology, Treatment, Biomarkers and Epigenetic Changes

Sugino

Sawada

Nakamura

2021

IJMS

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IgA, previously called Henoch-Schönlein vasculitis, is an essential immune component that drives the host immune response to the external environment. As IgA has the unique characteristic of a flexible response to broad types of microorganisms, it sometimes causes an autoreactive response in the host human body. IgA vasculitis and related organ dysfunction are representative IgA-mediated autoimmune diseases; bacterial and viral infections often trigger IgA vasculitis. Recent drug developments and the presence of COVID-19 have revealed that these agents can also trigger IgA vasculitis. These findings provide a novel understanding of the pathogenesis of IgA vasculitis. In this review, we focus on the characteristics of IgA and symptoms of IgA vasculitis and other organ dysfunction. We also mention the therapeutic approach, biomarkers, novel triggers for IgA vasculitis, and epigenetic modifications in patients with IgA vasculitis.

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“…However, it is thought to occur secondary to upper respiratory tract infections, drugs, vaccines, or malignancies [5]. Some pathogens known to be associated with IgAV are Streptococcus [6], S. aureus [7], Helicobacter pylori [8], varicellazoster virus [9], hepatitis virus [10], Parvovirus [11], human immunodeficiency virus [12], cytomegalovirus [13], and Clostridium difficile [14]. Due to the accumulation of IgA after an infection in small vessels and the presence of proinflammatory cytokines in the acute stage; recognized as postinfectious immune-mediated small vessel vasculitis [15].…”

Section: Introductionmentioning

confidence: 99%

Evaluation of the Effect of the Pandemic Period on Henoch-Schönlein Purpura

Çoban,

Ulu,

Sözeri

2024

Preprint

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Background Immunoglobulin A vasculitis is a vasculitis that occurs after a triggering infection and progresses with different clinical manifestations. Recent studies have shown an association between coronavirus disease and IgA vasculitis in children. In this study, it was aimed to evaluate the effect of the pandemic period on the course of IgA vasculitis. Methods Clinical data of 224 patients who were newly diagnosed with IgA vasculitis were retrospectively reviewed and grouped into prepandemic, pandemic and postpandemic periods. The clinical data of the patients in these periods were compared. Results In the prepandemic period, pandemic period and postpandemic period the number of patients were 75 [33.5%], 21 [9.4%] and 128 [57.1%], respectively. There was no significant difference between the periods in terms of isolated skin/systemic, gastrointestinal system and renal involvement [p>0.05]. Patients admitted with the diagnosis of IgA vasculitis in the prepandemic, pandemic and postpandemic periods comprised 0.5%, 0.2% and 0.3% of all patients, respectively. Conclusion While the number of patients admitted during the pandemic period decreased by 50%, it increased by 50% in the postpandemic period. It was thought that the reducing spread of viral infections with isolation measures may contribute to the decrease in IgA vasculitis cases.

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Vasculitis IgA como forma de presentación de la infección por el virus de inmunodeficiencia humana

Cited by 6 publications

References 6 publications

Henoch-schonlein purpura vasculitis revealing human immunodeficiency virus infection.

Henoch-schonlein purpura vasculitis revealing human immunodeficiency virus infection.

IgA Vasculitis: Etiology, Treatment, Biomarkers and Epigenetic Changes

Evaluation of the Effect of the Pandemic Period on Henoch-Schönlein Purpura

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