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“…Anemia is extremely common in dialysis patients and may have prompted workup for sources of gastrointestinal blood loss (26). Uremic platelet dysfunction combined with anticoagulation given during hemodialysis results in a bleeding diathesis that may bring gastrointestinal lesions to attention earlier (27). For instance, dialysis patients are more likely to have positive stool guaiac tests than nonuremic control subjects (28).…”
Section: Discussionmentioning
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“…Anemia is extremely common in dialysis patients and may have prompted workup for sources of gastrointestinal blood loss (26). Uremic platelet dysfunction combined with anticoagulation given during hemodialysis results in a bleeding diathesis that may bring gastrointestinal lesions to attention earlier (27). For instance, dialysis patients are more likely to have positive stool guaiac tests than nonuremic control subjects (28).…”
Section: Discussionmentioning
“…Correction of anemia with transfusion was shown to decrease uremic bleeding, and this treatment has therefore been supplanted with recombinant erythropoietin therapy. 21 Cryoprecipitate decreases bleeding time and bleeding symptoms for up to 24-36 hours, 22,23 so its use has generally been replaced with the vasopressin derivative 1-deamino-8-D-arginine vasopressin (DDAVP or desmopressin), which decreases clinical bleeding and improves measures of platelet function. 23,24 The effect of this treatment on bleeding time lasts for up to 4-8 hours.…”
Section: Renal Failurementioning
“…21 Cryoprecipitate decreases bleeding time and bleeding symptoms for up to 24-36 hours, 22,23 so its use has generally been replaced with the vasopressin derivative 1-deamino-8-D-arginine vasopressin (DDAVP or desmopressin), which decreases clinical bleeding and improves measures of platelet function. 23,24 The effect of this treatment on bleeding time lasts for up to 4-8 hours. However, neither correction of the bleeding time nor PFA closure times has been shown to be correlated with decreased uremic bleeding.…”
Section: Renal Failurementioning
“…Aspirin in this group of patients is an attractive potential primary prevention therapy because CVD risk is excessive compared to non-CKD individuals. However, caution has been given due to the higher risk of haemorrhage due to uraemia related platelet dysfunction [20]. It is suggested that aspirin-induced thromboxane inhibition-mediated impairment of platelet aggregation, combined with uraemia could lead to excess bleeding events.…”
Section: Discussionmentioning