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Cited by 3 publications
(3 citation statements)
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References 7 publications
(13 reference statements)
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“…Tumor suppressor function of cellular senescence has been identified in the context of the irreversible cell cycle exit 118 as well as autocrine and paracrine SASP mediated effects: cell cycle arrest reinforcement 119 122 propagation of senescent phenotype on neighboring damaged cells 123 and supporting tissue regeneration 124 126 . Moreover, SASP can elicit immune surveillance and clearance of both tumor 127 129 and neighboring damaged cells 124 .…”
Section: Discussionmentioning
confidence: 99%
“…Tumor suppressor function of cellular senescence has been identified in the context of the irreversible cell cycle exit 118 as well as autocrine and paracrine SASP mediated effects: cell cycle arrest reinforcement 119 122 propagation of senescent phenotype on neighboring damaged cells 123 and supporting tissue regeneration 124 126 . Moreover, SASP can elicit immune surveillance and clearance of both tumor 127 129 and neighboring damaged cells 124 .…”
Section: Discussionmentioning
confidence: 99%
“…Most of these studies were performed in the context of obesity-associated HCC in animal models without significant fibrosis. The discrepancy in these studies could be explained by differences in the senescence triggers, the composition of microenvironment, and extent of senescence surveillance in different animal models [48,50].…”
Section: Discussionmentioning
confidence: 98%
“…With the development of the disease, the fibrous tissue of the liver increases, which evolves into liver focal nodular hyperplasia—cirrhosis—liver cancer. Hypofunction of liver will seriously shorten the survival time of patients and even endanger the life safety of patients [ 23 ]. Therefore, the early diagnosis and treatment of liver cancer are very important for prolonging the survival time and improving the life quality of patients.…”
Section: Discussionmentioning
confidence: 99%